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Which of the following are NOT functions of the spinal cord?

maksim [4K]

Answer:

Reflexes are not a function of the spinal cord.

Explanation:

The spinal cord is part of the central nervous system and is made up of neurons that go along it (from the end of the brain stem almost to the bottom of the spine).

The spinal cord receives incoming messages from the peripheral nervous system (including the sensory neurons) and pass on messages from the brain to efferent neurons (motor neurons at muscles).

The spinal cord contributes to learning as it passes on the info received from sensory neurons (that there is an obstacle in the path) to the brain and passes on the response from the brain to the muscles (to lift the foot up higher).

The spinal cord is essential for integration as it passes on sensory information to the brain to determine motor output.

Reflexes are when sensory information is quickly sent straight to motor neurons to move. It does not need to go to the brain (hence does not need to go to the spinal cord) for the reaction as it would be too slow. This is seen when someone who is paraplegic (paralysed at lower torso) can still move their leg in response to the doctor tapping their knee even when they cannot forcibly move their legs.

Therefore reflexes are not a function of the spinal cord.

4 0

3 years ago

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A genetic mutation that does not result in a change in the amino acid sequence of the resulting protein is called A. a nonsense

kakasveta [241]

The answer on study island is "A SILENT MUTATION"

6 0

3 years ago

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Which term best demonstrates when an organism is better adapted to the environment that they live in and are most likely to surv

Maksim231197 [3]

The answer is natural selection. :)

7 0

3 years ago

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In a population of Mendel's garden peas, the frequency of the dominant A (yellow flower) allele is 80%. Let p represent the freq

DENIUS [597]

Answer:

If the frequency of the dominant allele in the pea population is 0.8, the genotype frequencies in the population would be 0.64 AA, 0.32 Aa, and 0.04 aa.

Explanation:

Hardy-Weinberg equation tells us that:

p2 + 2pq + q2

p2 is the frequency of AA plants

2pq is the frequency of Aa plants, and

q2 the frequency of aa individuals.

A allele in the population is 80%, the frequency is 0.8.

p represent the frequency of A allele. p is 0.8

Therefore calculating for q ( frequency of a allele).

p + q = 1.0

q = 1.0 − p

q = 1.0 − 0.8

q = 0.2

p = 0.8, q = 0.2.

Now we can calculate the predicted frequencies of the different genotypes, remembering that p2 is the frequency of the AA genotype, 2pq is the frequency of the Aa genotype, and q2 the frequency of aa genotype.

p2 = 0.8 x 0.8 = 0.64

2pq = 2 x 0.8 x 0.2 = 0.32

q2 = 0.2 x 0.2 = 0.04.

Thus, we would expect to see genotype frequencies of 0.64 AA, 0.32 Aa, and 0.04 aa in the pea plants.

6 0

3 years ago

Haase D, Germing U, Schanz J, et al. New insights into the prognostic impact of the karyotype in MDS and correlation with subtyp

vivado [14]

We have produced a huge, novel data set that incorporates morphologic, clinical, cytogenetic, and follow-up information from 2124 patients with myelodysplastic conditions (MDSs) at 4 organizations in Austria and 4 in Germany. Cytogenetic examinations were effectively acted in 2072 (97.6%) patients, uncovering clonal anomalies in 1084 (52.3%) patients.

Numeric and underlying chromosomal irregularities were reported for every patient and partitioned further as indicated by the quantity of extra anomalies. Along these lines, 684 unique cytogenetic classes were distinguished. The effect of the karyotype on the normal flow of the illness was concentrated on in 1286 patients treated with steady consideration as it were.

Middle endurance was 53.4 months for patients with ordinary karyotypes (n = 612) and 8.7 months for those with complex inconsistencies (n = 166). A sum of 13 uncommon irregularities were related to great (+1/+1q, t(1q), t(7q), del(9q), del(12p), chromosome 15 inconsistencies, t(17q), monosomy 21, trisomy 21, and - X), middle (del(11q), chromosome 19 peculiarities), or poor (t(5q)) prognostic effect, separately.

The prognostic significance of extra anomalies differed significantly relying upon the chromosomes impacted. For all World Health Organization (WHO) and French-American-British (FAB) order framework subtypes, the karyotype gave extra prognostic data. Our examinations offer new experiences into the prognostic meaning of uncommon chromosomal irregularities and explicit karyotypic mixes in MDS.

To learn more about prognostic impact of the karyotype in MDS from the given link

brainly.com/question/28189857

#SPJ4

7 0

1 year ago