Answer:
B) a nonsense mutation; this is because a nonsense mutation results in the change of a regular amino acid codon into a stop codon, which ceases translation. This fits with the problem's description of the protein that causes the symptoms as too short, as translation is the process by which proteins/polypeptides are created. A missense mutation would not be the answer because it still codes for an amino acid, which would not shorten the protein. A duplication of the gene would probably just lengthen the protein or not affect its length at all.
The DNA replicates it’s information in a process involving many enzymes. The DNA code for production of messenger is RNA. I’m eukaryotic cells, the MRNA is processed and migrates from the nucleus to the cytoplasm.
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Answer:
b. A transferase deficiency will result in an accumulation of the toxic metabolite galactosse 1-phosphate.
c. A galactokinase deficiency will cause an accumulation of galactose.
Explanation:
Transferase is an enzyme which is responsible for the breakdown of galactose which is a known milk sugar. Its deficiency causes the formation of toxic materials such as galactose-1-phosphate which comes from galactose, and galactitol. Galactokinase is also an enzyme which helps in the conversion of galactose into galactose 1-phosphate with the expenditure of ATP molecule, so its deficiency causes the deposition of galactose.
If a planet exists near a distant star, the star may have a very slight back and forth motion.
Water molecules are not found in a dna molecule.
