Prader-Willi syndrome (PWS) is a gentic disorder which has an impact on numerous physiological systems. PWS affected individuals (specifically babies) experience delayed growth, significant hypotonia (low muscle tone), and feeding issues. It does effect circadian rhythms in mice models.
SNORD116, often referred to as HBII-85, is a non-coding RNA (ncRNA) molecule that contributes to the alteration of other small nuclear RNAs (snRNAs). Unlike the majority of other snoRNAs, SNORD116 is not significantly complementary to ribosomal RNA and is expressed widely in the brain (but not in PWS patients).
According to the studies, SNORD116 cause sleep defect in patients with Prader-Willi syndrome. Same observation was seen in mouse models too. Paternal expression of SNORD116 is thought to be a potential gene for the sleep disruptions/circadian rhythm’s that the majority of PWS sufferers.
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ok, with what I don't see anything upload please
Answer:
AB
Explanation:
Blood group is discovered by famous scientist Karl Landsteiner in the year 1990. Co-dominance is the expression of the two alleles in the heterozygotes. In AB type, the two different alleles are present and they are expressed.
However in ABO type, it is govern by three allele namely A, B and O type. If only the antigen A is present, it is called A blood type. When only B antigen is present it is called B blood type and when both A and B are present, it is called AB blood type. And if neither of the antigen are present, it is called the O blood type.
The allele O is of recessive to both the blood type A and B. So a person with O blood type have two copies of O allele. But however A and B blood type are dominant of O, a person with A blood type may have one of the two genotypes -- AO or AA. Similarly, with blood type B, the genotypes are BO or BB.
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