Answer:
Explanation:
Galactosemia is an inherited metabolic disorder caused by changes, or mutations, in both copies of an individual’s GALT gene. In the majority of cases, children with galactosemia do not have parents with galactosemia. Mother and father separately are a silent carrier of the condition, which is expressed in the child (autosomal recessive inheritance).
Each individual has two copies of the GALT gene – one from father and one from mother. In case of any changes of these genes (mutations) that prevents the gene from working correctly. In order to inherit galactosemia, it is necessary for a child to have two GALT gene changes. In case of one GALT gene change there is no galactosemia.
Infants with galactosemia may be identified through newborn screening programs or by symptoms that present during the first few weeks of life. Untreated infants develop liver and kidney disease, cataracts in their eyes, and serious infections.
I Think The answer is d hope it helps
<em>In the inner medulla, urea recirculates, creating a greater osmotic gradient. It follows the concentration gradient formed by ADH-sensitive water reabsorption into the interstitial space from the collecting duct. The remaining water is drawn from the descending structures by increased interstitial osmolarity. </em>
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<em>To maintain the vertical osmotic gradient, the blood supply to the renal medulla serves as a countercurrent exchanger. - The medullary vertical osmotic gradient is maintained by the vasa recta.</em>
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<em>It enhances both water and urea permeability in the inner medullary collecting ducts, allowing urea to diffuse passively down its concentration gradient into the interstitial fluid. This contributes to the osmotic gradient and aids water absorption.</em>
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Answer:
I would have to say B is correct.
Explanation:
in a food pyramid the base is always producers.
Mitochondria, the power house of the cell