A DNA sequence that is 15 nucleotides long would normally give rise to a polypeptide sequence that would be 5 amino acids long. This is assuming that all the nucleotides in the DNA sequences are strictly coded to form only sense codons and not a single nonsense codon, also called termination codon
A sense codon is a set of three nucleotides also called a triplet, that codes for a particular amino acid. A DNA sequence of 15 nucleotides has 5 codons.
A nonsense or termination codon is one that does not code for any amino acid. There are three nonsense codons found on mRNA, and these are UAA, UAG and UGA. So if the DNA sequence has one of these, then the amino acids in the polypeptide chain will be 4 in number
The third one is the right answer.
Mutations appear randomly and these mutations can either have possitive, negative or neutral impact on the individual's survival. The environment of the individual amongst other factor determine the net effect of the mutation. For example:
A mutation on a shade plant for bigger leaves would have a possitive impact on the plants survival due to the increase of the photosynthetic tissue area. On the other hand the same mutation on a desert plant would have a negative impact on the plant's survival due to the increase of the leaf area which leads to increased water evaporation off the plant.
Yes considering tornadoes are formed by certain global temperatures.
This would depend on if the other parents has it or not. If they do not, they have a 50/50 chance that the trait will become recessive. That said, it could still be passed on to the child's kids in later generations.
The "Spanish" influenza pandemic of 1918–1919, which caused ≈50 million deaths worldwide, remains an ominous warning to public health. Many questions about its origins, its unusual epidemiologic features, and the basis of its pathogenicity remain unanswered. The public health implications of the pandemic therefore remain in doubt even as we now grapple with the feared emergence of a pandemic caused by H5N1 or other virus. However, new information about the 1918 virus is emerging, for example, sequencing of the entire genome from archival autopsy tissues. But, the viral genome alone is unlikely to provide answers to some critical questions. Understanding the 1918 pandemic and its implications for future pandemics requires careful experimentation and in-depth historical analysis.
