The menstrual cycle is complex and controlled by many different glands and the hormones that these glands produce.
The four(4) phases of the menstrual cycle are:
(1) menstruation.
(2) the follicular phase.
(3) ovulation .
(4)the luteal phase.
Common menstrual problems include heavy or painful periods and premenstrual syndrome (PMS).
Answer: DF508 mutation. A Genetic, Hereditary, Autosomal and Recessive Mutation.
Explanation:
Cystic fibrosis (CF) is a recessive autosomal lethal disease, it is most common on Caucasoid populations. Its diagnosis is suggested by the clinical features of chronic obstructive pulmonary disease, persistent pulmonary colonization (particularly with mucoid Pseudomonas strains), meconium ileus, pancreatic insufficiency with or familiarity history of the disease. The FC gene is large, with about 250 Kb of genomic DNA, 27 exons representing about 5% of genomic DNA; encodes a 6.5 kb transcribed mRNA. This mRNA is transcribed into a protein of 1480 amino acid called CFTR (Regulator Transmembrane Conductance Cystic Fibrosis). When a three-base pair deletion, adenosine-thymine-thymine (ATT) identified in the CFTR gene, exon 10, it results in the loss of a single amino acid phenylalanine at position 508 of the protein. This mutation is called DF508; “D” stands for deletion and “F” for phenylalanine amino acid.
Answer:
The advantages of selective breeding can include improved quality and higher ... breeding are positive for humans but an awareness of the disadvantages can help
Explanation:
A child presents with bone deficiency and a bowlegged appearance. This assessment data supports a diagnosis of:
a) Rickets
b) Osteoporosis
c) Osteomalacia
d) Paget disease
Answer:
a) Rickets
Explanation:
Rickets occurs at the early stage of childhood due to inadequate vitamin D.
Vitamin D provides the body with higher absorption rate of calcium and phosphorus, inadequate intake of Vitamin D results into low calcium and phosphorus levels in the bones, as a result, rickets sets in.
Rickets is a disarrangement of impaired mineralization of cartilaginous growth plates in children. It can also result from lack of exposure to sunlight.
