Answer: The parents of the white cat would be a dominant white cat and a recessive colored coat cat. The parents of the autosomal tabby would be a dominant tabby and either a recessive brown cat, striped cat, or a recessive black cat.
Explanation:
The genotypes and phenotypes would be : WW- Dominant white and ww- recessive color coat cat; their offspring would be 100% recessive white kittens
TT- dominant tabby and a recessive brown cat; their children would be 100%- dominant brown tabby
TT- dominant tabby and a recessive striped cat; children would be 100%- brown striped
TT- dominant tabby and a recessive black cat; children would be 100%- tabby kittens
The homology of animals is based on scientists current understanding on the evolutionary history of living species.
I think it's the nucleus. Isn't the nucleus the brain of the cell?
Answer:
- Calcium binds to troponin C
- Troponin T moves tropomyosin and unblocks the binding sites
- Myosin heads join to the actin forming cross-bridges
- ATP turns into ADP and inorganic phosphate and releases energy
- The energy is used to impulse myofilaments slide producing a power stroke
- ADP is released and a new ATP joins the myosin heads and breaks the bindings to the actin filament
- ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, starting a new cycle
- Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
Explanation:
In rest, the tropomyosin inhibits the attraction strengths between myosin and actin filaments. Contraction initiates when an action potential depolarizes the inner portion of the muscle fiber. Calcium channels activate in the T tubules membrane, releasing <u>calcium into the sarcolemma.</u> At this point, tropomyosin is obstructing binding sites for myosin on the thin filament. When calcium binds to troponin C, troponin T alters the tropomyosin position by moving it and unblocking the binding sites. Myosin heads join to the uncovered actin-binding points forming cross-bridges, and while doing so, ATP turns into ADP and inorganic phosphate, which is released. Myofilaments slide impulsed by chemical energy collected in myosin heads, producing a power stroke. The power stroke initiates when the myosin cross-bridge binds to actin. As they slide, ADP molecules are released. A new ATP links to myosin heads and breaks the bindings to the actin filament. Then ATP splits into ADP and phosphate, and the energy produced is accumulated in the myosin heads, which starts a new binding cycle to actin. Finally, Z-bands are pulled toward each other, shortening the sarcomere and the I-band, producing muscle fiber contraction.
