(705/224) / (224/224)
224/224 = 1.
So (705/224) / (224/224) = (705/224) / (1/1).
Dividing 2 fractions is equal to multiplying the first fraction by the inverse of the second:
(705/224) / (1/1) = (705/224) * (1/1) = 705/224.
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Answer:
An important function of seed dormancy is delayed germination, which allows dispersal and prevents simultaneous germination of all seeds. ... Many species of plants have seeds that delay germination for many months or years, and some seeds can remain in the soil seed bank for more than 50 years before germination.
Pituitary gland.
When you feel stress, the pituitary gland, at the base of the brain, increases its production of the hormone ACTH. This hormone tells the adrenal glands, found at the top of your kidneys, to increase their production of hormones. These stress hormones help you to concentrate, speed your reaction time, and boost your strength. Your hypothalamus also helps your body respond to stress.
Protein-protein interactions within the CARMA1-BCL10-MALT1 complex:
- The T-cell receptor and B-cell receptor-dependent NF-B induction and lymphocyte activation are mediated by the CBM complex, which is made up of the proteins CARMA1, BCL10, and MALT1.
- Each of the proto-oncoproteins CARMA1, BCL10, and MALT1 is a somatic gain-of-function mutation or chromosomal translocation, and dysregulation of CBM signaling is a characteristic of numerous lymphoid malignancies, including Activated B-cell Diffuse Large B-cell Lymphoma.
- Moreover, a number of immunological dysregulation diseases have been linked to both gain- and loss-of-function germline mutations in CBM complex proteins.
- Over the past ten years, careful examination of the interactions of CBM components has yielded a wealth of detailed structural knowledge.
- Here, we discuss important discoveries about the molecular nature of these protein-protein interactions that have helped the research develop a detailed understanding of how these proteins come together to form high-order filamentous CBM complexes.
- Approaches to therapeutic suppression of the CBM complex have thus far centered on obstructing MALT1 protease activity in order to treat lymphoid malignancy and/or autoimmunity.
- The structural effects of MALT1 protease inhibitors on significant protein-protein interactions are also reviewed in detail.
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