Well, you have to focus on the main part, all organisms need minerals (nutrients).... so, just think about the vitamins that are needed for the body....sorry if it didnt help you, i had a similar question a while ago....it helped me!
Answer:
Mitochondria and chloroplasts
Explanation:
These two organelles support the theory in that both have 70S ribosomes, the presence of a circular DNA within, and similar size. They can undergo division within the eukaryotic cyptoplasm. Both of these organelles are surrounded by a double membrane, which in the case of chloroplast is known to originate from ancient bacteria that lost its peptidoglycan component. Mitochondria and chloroplast both evolved from ancient free living bacteria that entered into larger cells, which in the case of mitochondria is known to be archaea and in the case of chloroplast is a eukaryotic cell. With this, these cells became dependent on it in a manner that it could not exist without it.
<h3>Hope this helps!</h3>
Cells break into 2’s and keep breaking apart
Answer:
1. Acetylcholine binds to receptors on the motor end plate
2. Ligand-gated channels open leading to depolarization
3. End plate potential triggers an action potential
4. Transverse tubules convey action potentials into the interior of the muscle fiber
5. Calcium is released from the sarcoplasmic reticulum
6. Calcium ions bind to troponin, which then moves tropomyosin
Explanation:
Acetylcholine (ACh) is a signaling molecule (neurotransmitter) that binds to receptors on muscle cells. This binding triggers the opening of ligand-gated sodium channels, thereby ions enter into muscle cells, which causes the depolarization of the sarcolemma and thus promotes the release of Ca2+ ions from the sarcoplasmic reticulum. The myoneural junction, also known as the motor endplate, is the site of synaptic contact between a motor axon and a skeletal muscle fiber. The endplate potential is the voltage that produces the depolarization of muscle fibers when ACh molecules bind to their receptors in the cell membrane. This depolarization spreads in the sarcolemma through transverse tubules (T tubules) and thus generates an action potential. Finally, this action potential induces the release of Ca2+ in the sarcoplasmic reticulum, which activates troponin protein and induces muscle contraction.
