An infant born with esophageal atresia and tracheoesophageal fistula receives a prescription for internal feedings after corrective surgery. <u>An infant is born with esophageal atresia and tracheoesophageal fistula.</u>
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Esophageal atresia is a beginning defect in which a part of a baby's esophagus (the tube that connects the mouth to the stomach) no longer increases well. Esophageal atresia is a start defect of the swallowing tube (esophagus) that connects the mouth to the belly.
The precise cause of EA remains unknown, but it appears to have some genetic additives. as much as 1/2 of all infants born with EA have one or greater other beginning defects, together with: trisomy thirteen, 18, or 21. other digestive tract problems, such as intestinal atresia or imperforate anus.
Oesophageal atresia is a concept to be because of trouble with the development of the esophagus even as the child is in the womb, although it's not clear exactly why this takes place. The condition is extra, not unusual in babies of mothers who had too much amniotic fluid in being pregnant (polyhydramnios).
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DIC is typically brought on by inflammation from an injury, disease, or infection. Typical causes include Sepsis: An inflammatory response to infection that affects the entire body. The most frequent risk factor for DIC is sepsis.
A dangerous condition known as diffused intravascular coagulation (DIC) occurs when the proteins that regulate blood clotting are overactive.
A combination of laboratory testing and clinical assessments are used to make the diagnosis of DIC. Low platelet count, increased D-dimer concentration, decreased fibrinogen concentration, and prolonged clotting times like prothrombin time are some laboratory findings that point to DIC.
The condition known as diffused intravascular coagulation (DIC) is characterized by an overactive state of the blood clotting proteins.
A combination of lab testing and clinical evaluations is used to diagnose DIC. Low platelet count, high D-dimer concentration, low fibrinogen concentration, and prolonged clotting times like prothrombin time are all signs of DIC in the lab (PT).
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Heart attacks happen suddenly when one of the arteries leading to the heart becomes blocked and cuts off the blood flow. Without oxygen, the heart muscles start to die.
Heart failure, on the other hand, usually develops gradually. The heart muscle becomes weaker and has trouble pumping blood to nourish the cells in your body. This is a chronic condition that gradually gets worse. But medications can help you live longer and better with it
Answer:
skeletal
circulatory
respiratory
digestive
immune
endocrine
nervous
muscular
Explanation:
skeletal- bones, spinal cord
circulatory- blood, vessels, heart,
respiratory- trachea, nose, lungs
digestive- intestines, stomach, oesophagus,mouth
immune- cells, organs, tissues
endocrine- kidney, bladder, urinary, glands
nervous- nerves, brain
muscular- muscle
