Answer:
If the question is referring to Mendel's postulates, all options are correct
Explanation:
Gregor Mendel discovered that certain components he called UNIT FACTORS determine inheritance. This unit factors were later described to be genes in modern genetics. He discovered that an organism receives two forms of this unit factors from each parent, which he called ALLELES. In his experiments, he observed that one of the pair of alleles have the ability to mask the expression of its variant pair. He called the allele that masks, DOMINANT allele while the allele that is masked, RECESSIVE allele.
During his cross experiment, he discovered that the alleles of a gene (unit factor) separates into gametes, he called this LAW OF SEGREGATION. He notably discovered in his cross involving two different characters that the segregation of the alleles of one gene into gametes does not influence the segregation of the alleles of the other gene. He termed this his LAW OF INDEPENDENT ASSORTMENT.
<span>Crust, mantle and core (core has 2 layers) </span>
Answer:
All cells are surrounded by a cell membrane. The cell membrane is semipermeable, allowing some substances to pass into the cell and blocking others.
Explanation:
Answer:
Employee boredom
Explanation:
When an employee specializes in a specific part of job, and it tends to be repetitive each day, it leads to Employee boredom
. Therefore, there should always be novel challenges and tasks in work place which do not make an employee feel boring about same monotonous work each day in which they are already specialized.
This never intends to mean that Job specialization is a bad thing, but there should be versatility in work because the work of repetitive nature tends to create a sense of boredom in person and makes his mind less creative as he is doing something daily on which he is expert and do not needs more learning.
Hope it helps!
Protein-protein interactions within the CARMA1-BCL10-MALT1 complex:
- The T-cell receptor and B-cell receptor-dependent NF-B induction and lymphocyte activation are mediated by the CBM complex, which is made up of the proteins CARMA1, BCL10, and MALT1.
- Each of the proto-oncoproteins CARMA1, BCL10, and MALT1 is a somatic gain-of-function mutation or chromosomal translocation, and dysregulation of CBM signaling is a characteristic of numerous lymphoid malignancies, including Activated B-cell Diffuse Large B-cell Lymphoma.
- Moreover, a number of immunological dysregulation diseases have been linked to both gain- and loss-of-function germline mutations in CBM complex proteins.
- Over the past ten years, careful examination of the interactions of CBM components has yielded a wealth of detailed structural knowledge.
- Here, we discuss important discoveries about the molecular nature of these protein-protein interactions that have helped the research develop a detailed understanding of how these proteins come together to form high-order filamentous CBM complexes.
- Approaches to therapeutic suppression of the CBM complex have thus far centered on obstructing MALT1 protease activity in order to treat lymphoid malignancy and/or autoimmunity.
- The structural effects of MALT1 protease inhibitors on significant protein-protein interactions are also reviewed in detail.
To learn more about protein-protein interaction visit:
brainly.com/question/14573382
#SPJ4
