D since gentics would make you more prone to drink and or have to drink less. same with age.
There are several systems in the body that get rid of cellular waste.
1. The urinary system removes urea from the body.
2. The spleen removes aged and damaged blood cells from the blood stream and breaks them down. The liver then removes the hemaglobin and breaks it down further. Some of the by products are excreted with the bile (ie heme) and some are released back to the blood stream to be removed by the kidneys (urea).
3. Some cellular waste is transported by the blood to the lungs where it is released through exhalation. (ie carbon dioxide)
<span>4. Some cellular waste is removed from the body through perspiration.
</span>Lysosomes have powerful enzymes and acids to digest and recycle cell<span> materials. Proteins aren't the only type of </span>cellular waste<span>. </span>Cells<span> also have to recycle compartments called organelles when they become old and worn out. For this task, they rely on an organelle called the lysosome, which works like a </span>cellular<span>stomach.</span>
Systemic lupus erythematosus (SLE) is a rheumatic disease attributed to autoimmune mechanisms.
Systemic lupus erythematosus (SLE, lupus) is characterized by a global loss of self-tolerance with activation of autoreactive T and B cells, leading to the production of pathogen autoantibodies and tissue damage.
Innate immune mechanisms are required for aberrant adaptive immune responses in SLE. Recent advances in basic and clinical biology have shed new light on the disease mechanisms of lupus. This review article describes recent studies that provide valuable insights into disease-specific therapeutic targets.
Systemic lupus erythematosus (SLE or lupus) is a systemic autoimmune disease with inflammation of multiple organs. SLE is characterized by the production of pathogenic autoantibodies against nucleic acids and their binding proteins, reflecting a global loss of self-tolerance.
Loss of tolerance with subsequent immune dysregulation is the result of genetic factors in the context of environmental triggers and stochastic events, with recent studies implicating more than 30 genetic loci in disease pathogenesis.
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