Answer:
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Explanation:
Answer:
<u>Proto-oncogenes</u>
- These genes code for protein that normally promote cell division
- Mutations that increase activity of these genes may lead to cancer
<u>Tumor suppressor genes</u>
- These genes code for protein that normally prevent uncontrolled cell division
- Some products of these genes normally function in repairing damaged DNA
- Mutation that decrease activity of these genes may lead to cancer.
Explanation:
<em>Proto-oncogenes</em> are group of genes that ordinarily help cells develop. At the point when a proto-oncogene mutates or there are such a large number of duplicates of it, it turns into a "terrible" quality that can turn out to be forever turned on or activated when it shouldn't be. At the point when this occurs, the cell becomes wild, which can prompt malignant growth. This terrible quality is called an oncogene.
Tumor suppressor genes are normal gene that hinder cell division, fix DNA missteps, or tell cell when to undergo apoptosis (die). At the point when tumor suppressor gene don't work appropriately or inactivated, cells can develop uncontrollable growth, that ultimately lead to cancer.
Explanation:
The lion (Panthera leo) is a large cat of the genus Panthera native to Africa and India.
Neurotransmitters can affect postsynaptic cells by causing molecular changes in the cells.
The term "chemical messengers of the body" is frequently used to describe neurotransmitters. The nervous system uses these molecules to send information between neurons or from neurons to muscles. A neurotransmitter influences a neuron in one of three ways: excitatory, inhibitory or modulatory.
When a stimulus is potent enough, the chemical messenger neurotransmitter is transferred into the nerve impulse. If it is powerful enough, it will reach the postsynaptic cell and affect the structure of the neuron. And when signal transduction happens, everything mentioned above is completed.
To know more about neurotransmitters visit the link:
brainly.com/question/15257730?referrer=searchResults
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