...that seemed to reflect the size of seeds the birds ate.
The answer would be "Behavior is modified as a cause"
Answer:
e. staphylococcal enterotoxin
Explanation:
Staphylococcal enterotoxin is a toxin that is produced by the bacteria Staphylococcus aureus. It is a toxin found inside the gastrointestinal tracts of the human body which is why it is called an enterotoxin and it results in food poisoning when consumed by humans.
Staphylococcal enterotoxin is proteinous in nature and it can be found on dairy products such as milk or cheeses that have been contaminated by Staphylococcus aureus.
After consumption of the contaminated milk or cheeses, the person begins to experience symptoms of nausea, diarrhea as well as vomiting.
Staphylococcal enterotoxin is a very heat stable toxin which means even though you heat up a food contaminated with this toxin, the bacteria may die by the toxins would still remain in the food.
The best way to avoid food poisoning by Staphylococcal enterotoxin is to avoid eating contaminated foods.
<span>is to regenerate the co enzyme NAD+, so that the glycolysis process can continue to breakdown glucose, produce ATP, and also produce pyruvate for the continuation of the process at the same time in anaerobic conditions when no oxygen is available.</span>
Answer:
a. Acetyl CoA carboxylase
Explanation:
Much of the fatty acids used by the body is supplied by the diet, excessive amounts of carbohydrates and protein obtained from the diet can be converted to fatty acids and stored as triglycerides. Fatty acid synthesis occurs mainly in the liver and mammary glands, and to a lesser extent in adipose tissue and kidney, the process incorporates acetyl CoA carbons into the forming fatty acid chain using ATP and NADPH.
The acetyl portion of acetyl CoA is transported to cytosol as citrate, produced by condensation of oxaloacetate and acetyl CoA, the first reaction of the citric acid cycle, this occurs when the concentration of mitochondrial citrate is high, observed when there is a high concentration of ATP and isocitrate dehydrogenase is inhibited. The increase of citrate and ATP favors the synthesis of fatty acids, since this pathway needs both. Acetyl CoA should be converted to malonyl CoA. Carboxylation is catalyzed by acetyl CoA carboxylase and requires ATP, this reaction is the regulated step in fatty acid synthesis: it is inactivated by products, malonyl CoA and palmitoyl CoA, and activated by citrate, another regulatory mechanism is reversible phosphorylation of enzyme, which makes it inactive due to the presence of adrenaline / glucagon
