Klinefelter's syndrome is a result of chromosomal nondisjunction mutations.
<h3>What is Klinefelter's syndrome?</h3>
Klinefelter's syndrome is a result of the presence of an extra X chromosome in germinal cells.
In genetics, this type of mutation is referred to as chromosomal nondisjunction (aneuploidies).
In conclusion, Klinefelter's syndrome is due to chromosomal nondisjunction.
Learn more about chromosomal nondisjunction here:
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Answer:
No
Explanation:
During cyclic photophosphorylation, electrons are cycled continuously out of and back into the reaction center of PSI. It does not involve PSII and plastoquinone. During non cyclic photophosphorylation, the reaction center of plant photosystem II passes electrons to plastoquinone. The reduced plastoquinone carries electrons to the cytochrome b6f complex. Since PSII and plastoquinones are not included in cyclic photophosphorylation, dichlorophenyldimethylurea would not affect the process.
Answer:
The mutation could cause serious damage in the replication process.
Explanation:
The TATA box is a DNA region that indicates the point where, processes like replication can begin. The mutation of this could cause that replication never begins, and therefore, protein synthesis is stopped. This severe complication would lead the cell to death in a short amount of time, for example.