Natural variation provided the phenotypes that natural selection then acted on, over time causing evolution
It has two phosppholipid layers that are either hydrophobic and hydrophillic the function is to protect thing from coming in and out
1. This process is called translation
2. This process is called transcription
I hope this helps :)
Answer:
<em><u>I</u></em><em><u> </u></em><em><u>HOPE</u></em><em><u> </u></em><em><u>I</u></em><em><u> </u></em><em><u>HELP</u></em><em><u> </u></em><em><u>YOU</u></em><em><u> </u></em><em><u>A</u></em><em><u> </u></em><em><u>LOT</u></em><em><u> </u></em><em><u> </u></em><em><u>.</u></em><em><u> </u></em><em><u>.</u></em><em><u>.</u></em><em><u>.</u></em><em><u>.</u></em><em><u>.</u></em><em><u>.</u></em><em><u>.</u></em><em><u>.</u></em>
Answer:
the answer is A. E. coli B
Explanation:
The multiplicity of infection (MOI) refers to the ratio between the numbers of viruses used to infect <em>E. coli</em> cells and the numbers of these <em>E. coli </em>cells. Benzer carried out several experiments in order to define the gene in regard to function. Benzer observed that <em>E. coli </em>strains with point mutations could be classified into two (2) complementary classes regarding coinfection using the restrictive strain as the host. With regard to his experiments, Benzer observed that rII1 and rII2 mutants (rapid lysis mutants) are complementary when they produce progeny after coinfect E. coli K (where neither mutant can lyse the host by itself). The rII group of mutants studied by Benzer does not produce plaques on <em>E. coli</em> K strains that carry phage λ (lysogenic for λ), but they produce plaques on <em>E. coli</em> B strains. This study showed that rIIA and rIIB are different genes and/or cistrons in the rII region.