Answer:
The statement that is true about leukotrienes is that they create excess mucus in the airways (last option).
Explanation:
Leukotrienes are inflammatory mediators, derived from arachidonic acid, in whose presence occurs increased vascular permeability in tissues, leading to inflammatory processes.
The presence of leukotrienes has been related to the development of allergic reactions, mainly in the respiratory tract, producing edema in the mucosa, contraction of the smooth muscles of the airways and increased production of mucus
in the airways.
the other options are not correct because leukotrienes:
- <em>Produce contraction of the smooth musculature around the airway.
</em>
- <em>Are related to the production of bronchial asthma.
</em>
- <em>Are inflammatory (chemical) mediators. </em>
90% of water gets reabsorbed. When water passes by the large intestines, most of it gets reabsorbed there in order to maintain normal organ functioning. Water reabsorption also serves as a factor in maintaining homeostasis so as to prevent dehydration.
The problems the girl exhibits when eating fruit is most likely due to elevated levels of fructose-1-phosphate in liver cells
Define Hereditary fructose intolerance (HFI).
Fruit sugar fructose and its precursors cannot be digested in people with hereditary fructose intolerance (HFI). The accumulation of fructose-1-phosphate in the liver, kidney, and small intestine is caused by a lack of activity of the enzyme fructose-1-phosphate aldolase.
The patient has HFI, which is due to a mutation in aldolase B. Sucrose would still be cleaved by sucrase, thus it would not increase in the stool. Fructose would not be metabolized normally, therefore it would be elevated in the blood and urine. Aldolase B would not cleave fructose 1-phosphate, thus its levels would be elevated and the product, glyceraldehyde, would not be produced.
HFI patients frequently grow to dislike fruit and sweets intensely. After consuming fructose-containing foods, they may experience symptoms like excruciating stomach pain, vomiting, and low blood sugar.
To know more about Hereditary fructose intolerance use link below:;
brainly.com/question/13700940
#SPJ4
Answer:
Three different ways in which antiretroviral drugs can prevent the formation of new viruses are:
- Inhibiting the reverse transcriptase enzyme in both nucleoside analogues and non-nucleoside analogues.
- Enzymatic inhibition of integrase enzymes.
- Inhibiting entry by antagonizing CCR5 co-receptors.
Explanation:
Antiretroviral drugs are those used for the treatment of human immunodeficiency virus (HIV) infection, making the effect of the virus on the host cell is less and preventing the development of AIDS.
This type of drug has been classified, according to their mechanism of action, being grouped into several families of drugs. Three different ways by which antiretroviral drugs can prevent the formation of virus are:
- <em><u>Inhibition of reverse transcriptase enzyme</u></em><em>: this enzyme is a DNA polymerase capable of forming single strand viral RNA by a process that is reverse to normal DNA to RNA transcription. Its inhibition prevents the formation of viral particles in the host cell. They are divided into nucleoside analogs and non-nucleoside analogs.</em>
- <em><u>Enzymatic Inhibition of Integrase enzymes</u></em><em>: this type of enzyme is contained in the virus, and is capable of causing a retrotranscribed DNA to integrate into the host cell's DNA so that viral replication occurs. Its inhibition prevents the replication process.</em>
- <em><u>Entry inhibitors or antagonists CCR5 co-receptors</u></em><em>: CCr5 is a membrane protein, found on the surface of CD4 T lymphocytes, which facilitates the entry of the virus into the cell. The drug acts as a competitive inhibitor of the viral particle, preventing the entry and infection of the host cell.</em>
The most effective treatments against HIV infection involve the use of two or more types of antiretroviral drugs.
