The development of resistance to the disease has taken different courses. In Australia, the virus initially killed rabbits very quickly – about 4 days after infection. This gave little time for the infection to spread. However, a less virulent form of the virus then became prevalent there, which spread more effectively by being less lethal. In Europe, many rabbits are genetically resistant to the original virus that was spread. The survival rate of diseased rabbits has now increased to 35%, while in the 1950s it was near zero.[15]
Hares are not affected by Myxomatosis, but can act as vectors.<span>[16</span>
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Answer:
World Health Organisation
Explanation:
"WHO" stands for World Health Organisation. It's an specialized agency of the U.N that is concerned with international public health.
Answer:
someone who moved from a different country looking for job opportunities
Explanation:
Answer:
Delta binds to the Notch receptor and this binding produces the cleavage of its intracellular domain, which subsequently enters into the cell nucleus to bind with a repressor in order to activate the transcription
Explanation:
The Notch signaling pathway is initiated when Notch receptors on the cell surface bind to the Delta ligand, which activates Notch signaling in cells next to it. In the receiving cell, Delta–Notch binding triggers the cleavage of the Notch intracellular domain called Nic (intracellular Notch). Subsequently, Nic enters into the cell nucleus where it releases repression on Suppressor of Hairless (Su(H)) class transcription factors, thereby activating the transcription of target genes.
If the disease was cause by the dominant allele, every person with said gene would be affected with the disease. if the genetic allele is recessive, both parents can be carriers (hetero) without being affected. that makes it easier for the genes to keep being passed on.
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