Answer:
It's a great tree for small properties because you only need one to produce fruit. Pluots are similar fruits. ... The seeds from the cross-pollinated fruit produce a different type of tree that has some characteristics of both trees.
Explanation:
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Hypothesis is what you think will happen before the experiment, it isn't necessarily backed up by any sources or evidence. A scientific law is the observation of a phenomenon and a theory is the explanation both can be used together to back each other up.
The bald eagle and the black bear are tetrapods, which means that they both have four limbs along with the digits. This shows that they both are the descendants of the four-limbed ancestors. However, the limbs of the bear and the bald eagle have different functions. The forelimbs of both these organisms serves different purpose. Hence, their limbs are homologous organs. The homologous organs are those which have the same ancestry but the functions are different.
Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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