A medial moraine is a narrow band of rock debris which usually runs down the centre of the glacier. It forms from the merging of the lateral moraines of two glaciers and often occurs in regions that have been acted upon by a past ice age.
I am not sure how it is for med
Answer:
The three processes from left to right are:
<u>Replication</u> DNA <u>Trancription</u> RNA <u>Translation</u> Protein
Explanation:
The process in question in the diagram is called the central dogma of life which describes the flow of genetic information from DNA to RNA to Protein. The three processes involved are:
- DNA Replication
- Transcription
- Translation
DNA Replication:
DNA replication is the process by which DNA makes a copy of itself. Replication of DNA is semi-conservative. this means that each new helix is a combination of an old (parent) strands and a new (daughter strand). The parental strand is used as a template to generate a complementary daughter strand.
Transcription:
Transcription is the formation of an RNA transcript of the DNA template. This process yields a mRNA that is further used as a code to manufacture proteins in the process of translation.
Translation:
Translation decodes the mRNA formed in transcription to generate proteins with specific amino acid sequence.
<span> Animal cells will swell when they are placed in a hypotonic solution</span>
When a DNA sequence alteration results in a stop codon rather than a codon that specifies an amino acid, it is known as a nonsense mutation. This is rarely seen in 10% of patients with genetic disease.
<h3>What is nonsense mutation?</h3>
A nonsense mutation in a DNA sequence causes a premature stop codon, also known as a nonsense codon, in the transcribed mRNA as well as a shortened, ineffective, and typically nonfunctional protein product.
Because stop codons, also known as nonsense codons, signal the completion of protein synthesis rather than encoding for an amino acid, they are the source of the term "nonsense mutation."
Examples of illnesses for which nonsense mutations have been implicated as contributing factors include: Cystic fibrosis (produced by the G542X mutation in the cystic fibrosis transmembrane conductance regulator); (CFTR) Beta-globin (thalassemia) Hurler disease.
Learn more about nonsense mutation, here:
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