Answer:
This probably because they destroy a given character or give rise to a new one.
Explanation:
Alteration here are the permanent changes found in the DNA structure. And are totally inheritable. An example is the case of sickle cell anaemia and haemophilia. People with this anaemia trait hardly live and reproduce even though they do not show ill effects.
Therefore, this mutation alteration is harmful because they destroy a given character.
If there is too much of glycogen and fat stored in a cell can be toxic. It damages the organs and leads to diseases.
Explanation:
Glycogen is stored in the liver or muscle tissue, the diseases affect functioning of the liver, kidney and the muscles. These diseases are mainly caused by genetic enzyme that is inherited from both parents.
The glycogen storage diseases are Von Gierke disease, Pompe's disease, Cori's disease, Hers' disease. The symptoms of this glycogen storage diseases are Low blood sugar, An enlarged liver, A swollen belly, weak muscles.
It is a metabolic disorder caused by enzyme deficiencies affecting glycogen synthesis in muscles.
Answer:
as the question does not contains image i have already mentioned the link to image in ask for detail section therefore i will answer according to that image.
the answer is "B"
Explanation:
Because before crossing over but after duplication both chromosomes will have same alleles as GgDd therefore according to the image the answer will be option "B". as this option showed duplicated chromosomes.
In hemophilia A it’s caused by a mutation in the gene for factor VIII. Hemophilia B is a result in a deficiency in factor IX due to a mutation in its corresponding gene.
In both cases, it is a mutation in the DNA (the macromolecule).
Answer:
Mutation.
Explanation:
Mutation in the organism's DNA that is passed on to the offspring is that main cause of heritable phenotypic change in some members of a population that reproduces asexually. Repeated exposure of Plasmodium that is sensitive to artesunate strain to increasing dosage of artesunate led to the development of resistance in plasmodium because the genetic mutation occur in the plasmodium that provide resistance against artesunate.
