Yes, Avery, Mc Leod and Mc Carty do thought that genes may be involved in the transformation of non virulent rough Strains of <em>Diplococcus pneumoniae</em> to harmful smooth strained bacteria
<h3><u>Explanation:</u></h3>
Avery was a Canadian medical researcher who along with other two well known scientists of the contemporary time went for an experiment where he took two strains of bacteria Diplococcus pneumoniae - one is rough and nonvirulent and another is smooth and virulent. For a control run, he injected both the bacteria in separate mice and the expected result was there. Now as he injected heat killed smooth bacteria, the mice survived. But as he injected heat killed smooth bacteria with rough bacteria, although there was no organism which can kill the mice the mice died. And autopsy revealed the presence of live smooth bacteria in the lungs.
Thus they suspected something have gone from the dead smooth bacteria into the non virulent rough bacteria which lead to transformation of the rough bacteria to smooth ones. Thus, the experiment was carried on, which suspected role of genes in this transformation.
Answer:
The mutation is on chromosome/karyotype 8.
Providing long-term control over the body's internal conditions
Keeping conditions within a normal range
Adjusting the "set point" for body temperature based upon level of activity
The lack of access to the healthy food has caused the growth of Sebastian to slow down. In the case healthy food is provided to him, he can grow into the size he was genetically suited to. This process is known as the canalization. Canalization term is used as the measure of the ability of any given genotype to result in the same phenotype in-spite of the environmental variability.
