The most logical answer is D, because, we can infer that they belong to the same genus in taxonomy, but they are different species
The correct answer is the last statement.
If the regulatory serine is mutated to alanine, then acetyl-CoA carboxylase will get activated spontaneously and will produce malonyl-CoA. The increased concentrations of malonyl-CoA will obstruct the oxidation of fatty acids by preventing the entry of fatty acids into the mitochondria.
It is because the AMP-activated protein kinase phosphorylates the serine residues of acetyl-CoA carboxylase to inactivate it. If a mutation occurs in such residues, then the AMPL cannot phosphorylate acetyl-CoA carboxylase and this enzyme will get activated spontaneously.
In such a situation, there will be more than sufficient production of malonyl-CoA, which will inhibit the admittance of more fatty acid getting inside the mitochondria; this will indirectly prevent the oxidation of fatty acids.
Muscles connect to your<span> skeleton and they contract and </span>move the skeleton along.<span> The calcified bones of </span>your<span> skeleton also work with the circulatory </span>system<span>.
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Answer: Proteins
Explanation:
The process where ribosomes use the mRNA to make proteins is called translation.
