Well... if the predator would become extinct, the population of the hunted will grow and reproduce. There for it would increase.
Answer:
They can perform photosynthesis.
Explanation:
Autotrophs are organisms that can produce their own food, and heterotrophs are organisms that feed on other organisms. This means that the ameba and paramecium can eat only other microorganisms.
The euglena is not limited to this. Hypothetically speaking, if there were no other microorganisms around it (which is unlikely), the euglena would not die as long as it is exposed to sunlight. Thanks to chloroplasts, organelles that contain chlorophyll, it can perform photosynthesis - a process in which, with the help of sunlight, carbon dioxide, minerals, and water are used to synthesize food.
Answer:
B. Glucagon
Explanation:
Glucagon is a pancreatic hormone, secreted by the alpha cells of islets of Langerhans. Whenever the blood glucose level falls, glucagon is released to increase the blood glucose levels. This function of glucagon is quite opposite to the function of insulin and hence both are antagonistic hormones. Insulin reduces the blood glucose where as glucagon increases the blood glucose.
Glucoagon is large polypeptide of 29 amino acids. Since it helps in increasing the blood glucose homeostatic levels it is called as hyperglycemic hormone. It does so by stimulating certain processes such as:
- Stimulating Glycogenolysis i.e breakdown of glycogen to release more glucose from liver.
- Stimulating Gluconeogenesis i.e. synthesis of glucose from non-carbohydrate sources like proteins.
- Glucagon inhibits the process of glycogenesis i.e. synthesis of glycogen, the storage form of glucose.
Answer:
Isoniazid (INH) and rifampicin (RMP).
This is called MDR TB.
MDR TB is a particular type of drug resistant TB. It means that the TB bacteria that a person is infected with are resistant to two of the most important TB drugs, isoniazid (INH) and rifampicin (RMP). If bacteria are resistant to certain TB drugs this means that the drugs don’t work.
