This insulin has no peak action and does not cause a hypoglycemic reaction.
<h3>What is
insulin?</h3>
- Insulin is a peptide hormone generated by beta cells of the pancreatic islets and is encoded by the INS gene in humans.
- Its name is derived from the Latin insula, which means "island." It is regarded as the body's primary anabolic hormone.
- It promotes the uptake of glucose from the circulation into liver, fat, and skeletal muscle cells, which controls the metabolism of carbs, lipids, and protein.
- The ingested glucose is transformed in these tissues into either glycogen (through glycogenesis) or fats (triglycerides), or, in the case of the liver, both, via lipogenesis.
<h3>What is the insulin's source?</h3>
- The pancreas, an organ behind the stomach that produces the hormone insulin, is responsible for this.
- The pancreas contains specific regions known as the islets of Langerhans (the term insulin comes from the Latin insula that means island).
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Answer: The traditional medical record for inpatient care can include admission notes, on-service notes, progress notes (SOAP notes), preoperative notes, operative notes, postoperative notes, procedure notes, delivery notes, postpartum notes, and discharge notes.
Explanation:
Answer:
The correct answer is option B. Production of Intrinsic Factor.
Explanation:
The kidney is a vital organ of the excretory system that plays many roles in the body. Exerting or filtering toxins, drugs, and metabolic wastes. Regulating fluid balance and concentration of the solutes in water, producing renin and erythropoietin and regulating pH which is an acid-base balance.
Intrinsic factor is produced by the parietal cell of the stomach which is essential to the absorption of the vitamin B12 in the small intestine.
Thus, the correct answer is option B. Production of Intrinsic Factor.
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
Answer:
Sexual and drug substance use risks should be determined during a routine health history with every new patient and updated regularly during periodic health care.
Risk assessment helps to identify individuals at risk; support recommendations for HIV, STD, and hepatitis screening; and establish risk reduction education topics and strategies.
Risk assessment can help people who are already infected access treatment
and learn how to avoid transmitting HIV to others.
Explanation:
