Explanation:
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This is a type III hypersensitivity reaction mediated by immune complex deposits. Immune complexes are antigen-antibody (commonly IgG) complexes that are soluble and prone to deposition in multiple organs. Once immune complexes are deposited in an organ, neutrophils and macrophages will then attack the organ causing organ damage and eventually failure. Type III hypersensitivity reactions are characteristic in SLE and other autoimmune diseases such as rheumatoid arthritis, etc.
Other types are type I hypersensitivity which are mediated by mast cells and histamine with the involvement of IgE and this commonly happens in allergic reactions. Type II hypersensitivity is cytotoxic hypersensitivity wherein antibodies directly attack organs (not forming immune complexes). Type IV hypersensitivity (or cell-mediated toxicity) involves T-lymphocytes. This is a delayed type of hypersensitivity exemplified by reactions from <em>M. tuberculosis</em> bacilli in tuberculous disease.
<span>The physical characteristics of minerals include traits which are used to identify and describe mineral species. These traits include color, streak, luster, density, hardness, cleavage, fracture, tenacity, and crystal habit.</span>
Answer:
Mitochondrial inner membrane. (Ans. C)
Explanation:
Proteins of the electron transport chain present in the mitochondrial inner membrane. The electron transport chain at the inner mitochondrial membrane forms a proton gradient, which is driving the synthesis of ATP with the help of chemiosmosis.
Oxygen forms water by accepting electrons, and picking up protons, and it sits at the end of the ETC. In the absence of oxygen ETC will stop running, and chemiosmosis will no longer able to produce ATP. Cells are not able to carry out the reaction they need to function, and may die after a long period, in the absence of ATP.
