The correct answer is Glucagon was secreted by his pancreas before he ate the cookies because his blood glucose was low. Insulin was secreted after he ate the cookies because his blood glucose was high.
At the time of hunger, the blood glucose level decreases, and glucagon at that time, that is, the hormone discharged by the islets of Langerhans increases the level of sugars in the body. The stored glucose in the liver gets discharged due to the presence of glucagon, which eventually elevates the level of glucose in the blood.
In the given case, when Kai ate the cookies, the level of glucose in the blood got increased and even elevated from the normal level in the blood. At that time, the insulin hormone is discharged from the islets of Langerhans, which helps the cells of the body to absorb the molecules of glucose and thus lowers the concentration of glucose in the blood. Therefore, both the hormones help in monitoring the levels of glucose in the body.
Answer:
Thanks for you question. Your hypothesis suggests a linear relationship between serum Cholesterol levels and MI. This hypothesis seems to ignore the difference in the prevalence and effectiveness of LDL receptors in the FH patient.
FH patients who have inherited the mutation from both parents have very few LDL receptors in their blood and therefore almost no ability to pass the unused Cholesterol through the liver. FH patients who are heterozygous will have more LDL receptors although both will find Cholesterol removal problematic without the addition of a PCSK9 inhibitor.
In short, your hypothesis need to account for other factors that are in play.
Explanation:
Consider my case. I am a 64 year old male who has Heterozygous Familial Hypercholesterolemia. Before treatment at age 12 my Total cholesterol was 510 mg/dl. My genetic testing shows two mutations to the LDL Receptor gene with only one mutation being pathogenic. My first heart attack was at 47 and first stroke at 62. My current LDL is too low to detect with the use of a PCSK9 inhibitor (Repatha®).
<h2>Muscular strength</h2>
Explanation:
Muscles that are elongated while activated (i.e., eccentric muscle action) are stronger and require less energy (per unit of force) than muscles that are shortening (i.e., concentric contraction) or that remain at a constant length (i.e., isometric contraction)
stronger muscle contraction includes:
- Potassium accumulate in the sarcoplasm
- Increase in muscle belly circumference
- Increased stimulus frequency
- Increased requirement
- Lesser proportion of motor neurons to muscle fibers
weaker muscle contraction includes:
- Begin contraction with muscle already 50% contracted
- Circular arrangement of muscle fascicles
- Lower sarcoplasm pH
By process of elimination you can find the right answer. We know for a fact that carbon is found in all living organisms. That is why we talk about carbon based life forms. So the first statement cannot be true. You can figure out how many Valence electrons an element has by looking at the group number. The group number of carbon is 4 so the 2nd statement cannot be true. We know that carbon does bond with other elements because it is not a noble gas and therefore must be able to form compounds.
This means the answer must be they can form up to four covalent bonds
