Training specialists need to be well aware of the wide variety of information stored in electronic health records. For everyday practice, one needs to know how and when to pull up such documents such as patient demographics, medical diagnoses, and treatments. Knowing where different providers' orders are stored is also crucial, for knowing when a specific order will take effect. There's a lot more that goes into learning what an EHR does than just understanding its features - there's a whole science behind how these systems work.
Although the extent to which EHRs are beneficial for training specialists is still debated, it is known that they can help to minimize errors in clinical documentation and improve efficiency. This has been shown across multiple studies - some children hospitals have seen reduced medication discrepancies after implementing electronic health records. The completion of tasks, including filling laboratory orders and checking labs, also improved significantly when using modern technology during patient care rounds at a large research hospital in New York. At the same time, some experts argue that process-driven activities through these systems could reduce face-to-face interactions between doctors on team shifts with each other's patients on observation status, leading to
Answer:
50 because in cash carti we trust
Explanation:
Answer:
adherence to his medication regimen, inhalation therapy, and instruction about methods of conserving energy.
the whole test is on Quizlet .
Answer:
Sedative hypnotics include benzodiazepines, barbiturates, and various hypnotics. Benzodiazepines such as Ativan, Librium, Valium, and Xanax are anti-anxiety medications. They also increase drowsiness and help people sleep
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’
