Enzymes are biological catalysts. Catalysts lower the activation energy for reactions. The faster the rate enzymes speed up reactions by lowering activation energy
- <em>it's </em><em>letter</em><em> </em><em>C </em>
Explanation:
<em>because</em><em> </em><em>the </em><em>R </em><em>called </em><em>in </em><em>sperm </em><em>nuclei</em>
<em>hope</em><em> it</em><em> helps</em>
MycolicWhat are the components of acid alcohol?Prevent contamination of the smear by the stain pptWhat is the purpose of placing a filter paper on the glass slide?<span>1. Flood with carbolfuchsin. Heat until steam is given off for about 5 min
2. Allow to stand for more 5 min w/o heating. Remove the filter paper
3. Decolorize with acid alcohol while continously agitating the slide, then wash with water
4. Counterstain with methylen blue for 30 sec., wash with water then let it dry</span>Discuss the procedure in using ziehl-neelsen methodRedWhat is the color of an acid fast bacteria?Blue/greenWhat is the color of non-acid fast bacteria?<span>4g basic fuchsin
20 ml of 95% ethanol
8 ml phenol
100 ml distilled water</span>What are the components of kinyoun's carbolfuchsin?<span>1. Flood the smear with kinyoun's for about 5.min
2. Rinse with deionized water
3. Decolorize to faint pink color with acid alcohol for 3 min
4. Rinse again with deionized water
5. Flood with methylene blue for (4min), dry and examine under OIO</span>Discuss the procedure in using kinyoun's methodHorizontal or vertical scanningWhat is the proper way or direction of reading your stained smear?Horizontal scanningWhat is known as standard scanning?Pappenheims methodIn what method we use rosalic acid and alcohol as decolorizer?ColorlessWhat is the reaction of Mycobacterium smegmantis when rosalic acid and alcohol was added as decolorizer in pappenheims method?Diluted alcholic fuchsinWhat is use as stain i youe baumgarten's method?Red
1.photosynthesis is anabolic while cellular respiration is catabolic
2.photosynthesis occur in the day while cellular respiration take place any time
Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
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