The answer is (c) If the pH of the soil is acidic, then the plants will grow larger!
Hope it helps! ^^
Answer:
NEWS
What Happens to Tumor Cells After They Are Killed?
Oncology Times: December 25, 2017 - Volume 39 - Issue 24 - p 46-47
doi: 10.1097/01.COT.0000528040.85727.60
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tumor cells: tumor cells
Researchers from Harvard Medical School, Boston, and the Institute for Systems Biology, Seattle, have discovered that the remains of tumor cells killed by chemotherapy or other cancer treatments can actually stimulate tumor growth by inducing an inflammatory reaction. The study also reveals that a family of molecules called resolvins can suppress this unwanted inflammatory response, suggesting new ways to enhance the effectiveness of existing cancer therapies
Conventional, radiation- and drug-based cancer therapies aim to kill as many tumor cells as possible, but the debris left behind by dead and dying cancer cells can stimulate the production of proinflammatory cytokines, signaling molecules that are known to promote tumor growth.
“Dead and dying tumor cells are an underappreciated component of the tumor microenvironment that may promote tumor progression,” explained Charles N. Serhan, PhD, Director of the Center for Experimental Therapeutics and Reperfusion Injury at Brigham and Women's Hospital and Professor at Harvard Medical School. Serhan and colleagues therefore decided to investigate whether tumor cell debris can stimulate tumor growth.
Explanation:
Abnormally slow depolarization of the ventricles would most change the shape of the QRS complex in an ECG tracing.
- The conduction system in the ventricles depolarizes, and this depolarization spreads along the walls of the ventricles to produce the QRS complex.
- It represents electrical activity that occurs before the ventricles contract (ventricular systole). The QRS complex appears immediately after the onset of ventricular systole.
- An aberrant depolarization of the ventricles results in the formation of an abnormal QRS complex.
- The SA node, an ectopic pacemaker in the atria, AV junction, bundle branches, Purkinje network, or the ventricular myocardium are all possible pacemaker sites in these aberrant QRS complexes.
- An aberrant QRS complex might have any shape, from normal to wide and odd to slurred and notched.
learn more about QRS complex here: brainly.com/question/10842164
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Answer: If a mutation occurred in Silencer of Death Domain (SODD) that prohibited its interaction with the DD of TNF receptor, the TNF receptor would evoke its response by binding to a transmembrane receptor, TNFR1.
Explanation: Binding to TNFR1 inhibits the recruitment of cytoplasmic signaling proteins to TNFR1 to prevent recruitment of a number of protein (TRADD) or prevent spontaneous aggregation of the cytoplasmic death domains of TNFR1 molecules.
SODD mechanism is useful in preventing unwarranted TNFR1 activation in the process of apoptosis.
