Answer: 0.33ml
Explanation:
What you have: 1,200,000 units/2ml
What you need: 400,000 units/x
1,200,000/2ml x 400,000/x =
400000/1,200,000 = 0.33ml
check answer: 0.33333333 x 1,200,000= 400,000 units
Answer: True
Explanation:
The corticospinal tract can be defined as the white matter motor pathway that starts with the cerebral cortex and terminates at the lower motor neuron of the spinal cord. This controls the movement of the trunk and limbs.
If the corticospinal tract is affected at any level above the medulla the voluntary control over the movements will be affected on the contra-lateral side of the body.
Answer:
The answer are A, B and C
Explanation:
Skeletal traction is a temporary way of handling some types of fractures. It consists in the application of a force to a body part. Its use is to minimize muscle spasms, reduce, align and immobilize fractures, reduce deformities. Pulley lines and weights are used, which must be free of any obstacles, the weight should not rest on the bed. sheets must be changed daily for hygiene; and the patient's uncommitted foot should rest freely in the bed.
Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.
What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?
- Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
- To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
- 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
- At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.
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