Endotherms need to consume more food to keep their metabolism up and maintain consistent temperatures (homeotherapy). This is because they are a warm-blooded organism which is dependent on the internal generation of heat.
As per the demonstration of Griffith and Avery, it can be concluded that the given experiment finding supports hypothesis III, i.e. genetic material or transforming substance is DNA.
<h3>What is Genetic material?</h3>
Genetic material may be defined as any substance found in the cells of plants, animals, microbial or other origins that hold genetic information and that departs it from one generation to the next.
In 1944, Avery and others concluded that the transforming material was pure DNA, not protein or RNA.
These investigators found that DNA extracted from a virulent strain of the bacterium Pneumococcus genetically transformed an avirulent strain of the organism into a virulent form.
Therefore, as per the demonstration of Griffith and Avery, it can be concluded that the given experiment finding supports hypothesis III, i.e. genetic material or transforming substance is DNA.
To learn more about Griffith's experiment, refer to the link:
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Long-term potentiation (LTP) is considered a cellular correlate of learning and memory. The presence of G protein-activated inwardly rectifying K(+) (GIRK) channels near excitatory synapses on dendritic spines suggests their possible involvement in synaptic plasticity. However, whether activity-dependent regulation of channels affects excitatory synaptic plasticity is unknown. In a companion article we have reported activity-dependent regulation of GIRK channel density in cultured hippocampal neurons that requires activity oF receptors (NMDAR) and protein phosphatase-1 (PP1) and takes place within 15 min. In this study, we performed whole-cell recordings of cultured hippocampal neurons and found that NMDAR activation increases basal GIRK current and GIRK channel activation mediated by adenosine A(1) receptors, but not GABA(B) receptors. Given the similar involvement of NMDARs, adenosine receptors, and PP1 in depotentiation of LTP caused by low-frequency stimulation that immediately follows LTP-inducing high-frequency stimulation, we wondered whether NMDAR-induced increase in GIRK channel surface density and current may contribute to the molecular mechanisms underlying this specific depotentiation. Remarkably, GIRK2 null mutation or GIRK channel blockade abolishes depotentiation of LTP, demonstrating that GIRK channels are critical for depotentiation, one form of excitatory synaptic plasticity.
Learn more about receptors here:
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Answer:
True
Explanation:
Ans;true
The neural plate is one of the features exclusive to the embryonic development of chordates. It is located dorsally to the notochord. The neural plate is the rudimentary nervous system of the embryo. During early development ,the neural plate is formed from the ectodermal layer . It undergoes changes in the position and arrangement of it's cells to form the brain and the spinal cord which comprises the central nervous system. It forms tissues and eventually organs by the deepening of the groove which forms ridges which eventually folds and fuses to form the neural tube . The tube differentiates into the prosencephalon, the mesencephalon and the rhombencephalon, from which the organs are formed
Nucleotide!
When attached in a polymer, they become a nucleic acid.
