Letter M is in between L and N. So like this LMN and o is in between N and P. So like this NOP. So technically the difference from o to m is just one of N.
Answer: (c) It generates ATP, which cells can use to do work.
(d) It generates chemical gradients, which have potential energy.
Explanation:
The overall process of Cellular respiration is the aerobic break down of organic compounds (food) with the release of CO2, water and energy in form of ATP which drives most cellular work.
Organic compound + oxygen --------> carbon dioxide + water + energy (ATP + heat)
In cellular respiration during the break down of food in glycolysis and the citric acid cycle, chemical energy (NADH and FADH) are extracted from each cellular process and transported to the electron transport chain built in the inner mitochondrial membrane. The chemical energy harvested from food is transformed into a proton-motive force, a gradient of H+ across the membrane. When this H+ election fall from their gradient via ATP synthase, Chemiosmosis couples this proton motive force to phosphorylate ADP to form ATP.
Sucrose is a processed sugar so due to chemical composition it cant break down and also enzymes break down natural sugars not processed.
Answer:
commenalism
Explanation:
The salad is neither harmed, nor benefits from this
Answer:
Leptin is a protein hormone composed of 167 amino acids of the cytosine family, it is synthesized mainly by white adipose tissue. The leptin receptor is related to that of receptors for cytosines (since it uses Janus or JAK type kinases and STAT-3 proteins as intracellular mediators of transcription pathway), it is predominantly found in the hypothalamus (particularly in the arcuate nucleus and to a lesser extent in the nuclei dorsomedial and ventromedial), hippocampus and cerebellum. There is evidence that leptin acts at the level of the arcuate nucleus, preventing the formation of NPY.
Explanation:
Leptin is produced exclusively in adipose tissue cells in a wide variety of species, including humans, and its concentration is higher in overweight than in lean individuals. The biological actions of leptin can be classified into two groups, those that are exerted in the tissues of the central nervous system, mainly the hypothalamus, and those that are carried out on the peripheral tissues. The former regulate body weight downward, decrease food intake, increase basal energy expenditure and modify some neuroendocrine functions such as reproduction, while the latter have effects on proliferation, differentiation and metabolism of peripheral tissues. The leptin that reaches the hypothalamus, in addition, inhibits protein synthesis and the secretion of neurons producing NPY / AgRP of the arcuate nucleus and stimulates the synthesis and secretion of those containing POMC. NPY (neuropeptide Y) is produced in the hypothalamic arcuate nucleus. This neurotransmitter arrives through the projections of the neurons of said nucleus to the paraventricular nucleus, which is the area where it is released. There are several studies that have shown that an increase in NPY leads to an increase in the sensation of hunger and thus to hyperphagia and obesity. Leptin acts at the level of the arcuate nucleus, preventing the formation of NPY. The leptin receptor lacks enzymatic activity in its intracellular domain. Instead, it is linked to members of the janus kinase family, which belong to a class of tyrosine kinases. The binding of the ligand activates the Jak kinase and leads to the phosphorylation of certain cytoplasmic proteins. Within these proteins there is a class of cytoplasmic transcription factors called signal transducers and activators of transcription (STAT).
