True. it starts off simple then you move into details
The increase in insulin level following an increase in glucose level in the blood can best be explained by A FEED BACK MECHANISM THAT REGULATE THE BLOOD GLUCOSE LEVEL.
Insulin is an hormone that is secreted by the pancreas and it functions by regulating the amount of glucose that is found in the blood. When the level of glucose in the blood is higher than normal, insulin interfere by converting the excess glucose into glycogen. The glycogen is stored in the liver and in the skeletal muscles and they will only be converted back into glucose, if the level of glucose in the blood is lower than normal.
THE CELL-MATCHING Nome Kocelle Match the descriptions in Column with the name in Column Column ! - 1. holds nucleus together a. Golol bodies 2 surface for chemical activity b. nucleus 3. units of heredity ochromosomes 4. digestion center d. VOUI 5. where proteins are made o rosomes 6. structures involved in mitosis in animal cells only 1. endoplasmic reticulum 7. microscopic cylinders that support nucleor membrane and give the cell shape h controles 8. shapes and supports a plant coll cytoplasm 9. stores and releases chemicals chlorophy 10. food for plant cells is made here k chloroplasts 11. spherical body within nucleus 1. col (plasma) membrane 12. controls entry into and out of coll m. cell wall 13. fraps light and is used to produce n. mitochondria food for plants olysosome 14. chromosomes are found here p. genes 15. Jellylike substance within cell a nuclear pore 16. contains code which guides all cell nucleolus activities S. plastid 17. minute hole in nuclear membrane 18. "powerhouse of cell 19. contains water and dissolved minerals t. microtubule 20. stores food or contains pigment Biology 1F8765 cinstructional Fair,
Answer:
d. Ribosome
f. Cell wall
Explanation:
In humans 80s type of ribosome is present and in bacteria 70s type of ribosome is present. Human cells do not have cell wall while bacterial cells have peptidoglycan cell wall. These differences can be targeted by the potential antibacterial agents.
For example, tetracycline antibiotic inhibits the binding of important molecules to bacterial ribosome which ultimately inhibits the protein synthesis in bacteria. Vancomycin antibiotic on other hand inhibits the cell wall formation in bacteria by inhibiting peptidoglycan synthesis.
