Answer:
The correct answer is option a, that is, the thrM and thrN would be transcribed at high levels in comparison to non-mutant.
Explanation:
Based on the given information, in the existence of thorain the expression of thrM and thrN will be prevented as thorain functions as a negative regulator. In case, if the gene, which codes for thorain, that is, thrL gets mutated, the coding for the functional thorain would get prevented.
With the encoding of the non-functional thrL protein, there would be an enhancement in the expression of thrN and thrM genes and it will get transcribed at higher levels in comparison to the non-mutant or the wild-type.
Explanation:
Complete answer:
A mature erythrocyte lacks nucleus and mitochondria so as to make a place for the accommodation of more hemoglobin and hence more oxygen molecules. Lack of such organelles also provides the peculiar biconcave appearance of RBCs that aids in efficient diffusion. Young mammalian RBCs are nucleated.
The statement above is TRUE.
For patients, who recently received an autologous transfusion, they can not be reliably antigen typed. This is because, mixed field reaction typically occur when the phenotyping process in underway. The donor cells usually react as expected with the typing serum but the patients' autologous cells do not react with the typing serum.
Answer: Circulatory system, Cardiovascular system, and Digestive system.
Explanation:
