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Tomtit [17]
3 years ago
10

Some cognitive neuroscientists study brain lesions to learn more about brain functions. However, a major problem with this techn

ique is that ____________.
Medicine
1 answer:
Fudgin [204]3 years ago
6 0

Explanation:

There are seven challenges for neuroscience:

BY THE DEFINITION/FROM A SOURCE:

Challenge 1. Change the way neuroscience is done.

Challenge 2. Data ladders.

Challenge 3. Predictive neuroscience.

Challenge 4. Simulating the brain.

Challenge 5. Classifying and simulating diseases of the brain.

Challenge 6. From the brain to brain-inspired technology.

Challenge 7. Working with society

Those are the challenges they have, and they each represent a different problem.

Brain lesions:

Nausea, vomiting, personality changes, headaches...

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A client is receiving somatropin. the nurse should monitor which most significant laboratory study during therapy with this medi
lapo4ka [179]

A client is receiving somatropin. the nurse should monitor <u>Thyroid-stimulating hormone level </u>

<u />

  • Somatropin injections are used to treat growth hormone insufficiency in both adults and children. Growth hormone is a natural hormone produced by your body.
  • Children with specific diseases that hinder normal growth and development can potentially benefit from somatropin injections to accelerate their growth.
  • The U.S. Food and Medicine Administration (FDA) has licensed the drug somatropin for a number of conditions, but it is mostly used to treat growth problems in children and growth hormone insufficiency in adults.
  • For the purpose of treating HIV-related cachexia or wasting in patients with the virus, somatropin is FDA-approved under the trade name Serostim.
  • Somatropin may be used off-label to treat the lipodystrophy syndrome linked to HIV.

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3 0
1 year ago
In a medical setting,what's done to whole numbers to minimize the chance of errors?
loris [4]
Ensure correct entry of the prescription. ...
Confirm that the prescription is correct and complete. ...
Beware of look-alike, soundalike drugs. ...
Be careful with zeros and abbreviations. ...
Organize the workplace. ...
Reduce distraction when possible.
6 0
3 years ago
Tsu LV, Dienes JE, Dager WE. Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose
lianna [129]

Subsequent INR readings are influenced by the dose, method, and initial INR of vitamin K. For intravenous vitamin K doses of 2 mg or more, INR decrease is comparable. FFP preadministration has no effect on INR readings 48 hours or more after vitamin K administration.

What is Abstract of Vitamin K dosing to reverse warfarin based on INR, route of administration, and home warfarin dose in the acute/critical care setting?

  • Commonly, vitamin K is used to reverse the anticoagulant effects of warfarin. The ideal vitamin K dosage and delivery method that does not lengthen bridging therapy are still unclear.
  • To ascertain the elements affecting the level and pace of vitamin K-induced INR reversal in the acute/critical care setting.
  • 400 patients' charts from between February 2008 and November 2010 who got vitamin K to counteract the effects of warfarin were examined. International normalized ratios (INRs), intravenous or oral vitamin K doses, and whether or not fresh frozen plasma (FFP) was administered were among the information gathered. INRs were measured 12, 24, and 48 hours before vitamin K treatment.
  • At baseline, 12 hours, 24 hours, and 48 hours, respectively, intravenous vitamin K decreased INR more quickly than oral vitamin K (5.09, 1.91, 1.54, and 1.41 vs. 5.67, 2.90, 2.14, and 1.58). Subsequent INR values were impacted by baseline INR (p 0.001), method of administration (p 0.001), and vitamin K dosage (p 0.001). For intravenous vitamin K doses of 2 mg or more, there was a similar drop in INR. Home warfarin dose had no effect on INR responses to intravenous or oral vitamin K (p = 0.98 and 0.27, respectively). FFP had no effect on INR readings 48 hours later. Although larger vitamin K doses and longer anticoagulation bridge therapy appeared to be related, neither the incidence (p = 0.63) nor the duration (p = 0.61) were statistically significant.

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7 0
2 years ago
Name and briefly describe the three functions of the small intestine.
NARA [144]

Answer:

Functions of the small intestine:

- Digestion process = contributes to end digestion by adding pancreatic and bile juices to the process.

- Absorption process = Aids in the absorption of water, vitamins, proteins, salts, fats necessary for the body.

- Food transit (peritalism) = the chyme formed in the stomach continues its process in the intestine where more digestive enzymes are joined until it is taken to the large intestine with many products already discarded from the digestion.

6 0
3 years ago
Even though there are a few more weeks of warm summer weather, Cecile's doctor at the Senior Community Center has recommended th
umka21 [38]

Answer:

The related cell-mediated immune system to the doctor's recommendation is the T-lymphocytes or T-cells

Explanation:

Immunity is defined as the body's ability to protect itself against invading harmful organisms or substances. There are two forms or immune responses by the body, and they include:

1. innate immunity (non-specific immunity): this type of immunity is the one present at birth, they include the skin barriers, phagocytes, mucous membranes, stomach acids (HCl), enzymes etc. This immune response is usually the first line of defense against all invading antigen, hence they are non-speciific.

2. acquired or specific immunity: this type of immune response is specific to an organism or group of organisms of the same species. Acquired immunity are of two types; passive and active

i. passive acquired immunity is gotten as a result of transfer from one organisms that is already immune to another organism. It is a temporal form of acquiring immunity, and it is most commonly gotten through transfer through the placenta, or the transfusion of blood or blood products, where the recipient is protected for weeks or even months.

ii. Active immunity is further divided into antibody- and cell-mediated immunity. In the anti-body mediated immunity, B-lymphocytes manufacture antibodies (chemical immune agents) against antigens and the memory cells store the synthesis pattern, so that for future re-infection, the antibodies are manufactured again.

Now, the second part is the cell-mediated active immunity which is specific to our question. This type of immunity is mediated by T (thymus) lymphocytes or T-cells. T-cells because they originated and grow in the thymus gland. T-cells are further divided into three chief types according to their functions:

a. T-helper cells: this type of T-cells stimulate the activation and activity of other cells to enhance immune response, for example, it stimulates B-cells to produce antibodies or phagocytic macrophages for phagocytosis

b. T-suppressor cells: this helps to down regulate the activities of the immune cells/response, by suppressing its activity. they are beneficial to prevent auto-immune responses, but are detrimental if active in cases of active infection.  

c. T-cytotoxic cells: these cells recognize and destroy infected cells, and also activate phagocytes for phagocytosis or ingested pathogens.

I will recommend that you go and look up pictorial representations of these immune responses and cells, they will help your understanding.

5 0
3 years ago
Read 2 more answers
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