They are like that because they can't always be faulty and may have at least some spark of truth to this theory also as stated earlier a theory in scientific terms is just an observation so it can't really be turned down hence theory.
Answer:
C U A G C U A U G G U A A G C C G C G U A U G A A G C
Explanation:
this is the complementary strand in mRNA.
A=U
G=C
C=G
T=A
this is the answer key to any mRNA strand.
Hope this helps! :)
Answer:
: todas las bacterias se pueden clasificar en una de las tres formas básicas: esferas (cocos), bastones (bacilos) y espirales o hélices (espiroquetas). Necesidad de oxígeno: las bacterias también se clasifican en dos grupos, según si necesitan oxígeno para vivir.
explicacion:
:-t
Answer:
telophase
Explanation:
because the chromatids are pulled to the opposite sides
Answer:
1. The difference between the normal hemoglobin protein DNA sequence and the sickle cell hemoglobin DNA sequence is a base to base shift, in this case adenine (GAG) to thymine (GTG).
2. The difference affects the amino acid sequence of the protein by replacing glutamic acid (Glu) with valine (Val).
Explanation:
In sickle cell anemia, a change in the DNA nucleotide sequence is observed, where adenine is substituted by thymine, whose expression is the change in the amino acid sequence of globine β, incorporating valine instead of glutamic acid. This represents a molecular mutation - point mutation - by subtitution, which corresponds to missense mutation.
<u>Normal hemoglobin protein in a RBC</u>
DNA CTG ACT CCT GAG GAG AAG TCT
Amino acids Leu Thr Pro Glu Glu Lys Ser
<u>Sickle cell hemoglobin protein in a RBC</u>
DNA CTG ACT CCT <em>GTG</em> GAG AAG TCT
Amino acids Leu Thr Pro <em>Val</em> Glu Lys Ser
When GAG is transcribed to mRNA, the CUC codon is obtained, which codes for glutamic acid. Thymine substitution causes the DNA sequence to change to GTG, which is transcribed as CAC, the codon that encodes the amino acid valine. The <u>change from glutamic acid to valine in β-globin causes an altered hemoglobin, giving the abnormal erythrocytes observed in sickle cell disease</u>.
