No it doesn’t matter as long as you are able to explain it
<h2>Answer:</h2><h2><em>About Two Thousand</em></h2><h2><em>About Twenty-Five Thousand</em></h2><h2><em>1780</em></h2><h2><em>About a Third Of</em></h2><h2>Explanation: </h2><h2><em>Thank Me Later</em></h2>
The probability of the mother to pass down an abnormal long chromosome 13 is of
. The probability of the father to pass down an abnormal short chromosome 11 is also of
.
When meiosis occurs in order to produce gametes, firstly there is the separation of the two homologous chromosomes, leading to the formation of two cells with only n chromosomes. After this first division, another division happens over these two newly formed cells segregating the two sisters chromatids of each chromosome into two new cells again. In the end, there are 4 gametes formed where 2 have a copy of the same chromosome and the other two of a copy of the homologous chromosome. So, when forming gametes, both this man and woman, would create half gametes with an abnormal chromosome.
So, the probability of producing an offspring that will have both a long chromosome 13 and a short chromosome 11 is found by multiplying both probabilities of each individual parent passing the abnormal chromosome.
×
=
The probability is of
or 25%.
If such a child is produced, the probability that this child would eventually pass both abnormal chromosomes to one of his or her offspring is of
as well. Going back to the meiosis, the possible four gametes that could result from one of this individual's meiosis were either 2 with both abnormal chromosomes and 2 with both normal chromosomes, if the chromosomes were segregated together to the same cell, or all 4 with an abnormal chromosome and a normal chromosome, if the chromosomes were segregated separately to different cells. Considering this 8 possible resulting cells and only 2 of them having both the abnormal chromosomes we may conclude that
=
is the probability that this child would eventually pass both abnormal chromosomes to one of his or her offspring.
A more recently developed cancer treatment is targeting telomerase activity. So the correct answer is b)stimulating telomerase activity.
A topic of ongoing attention is the use of telomeres and telomerase as potential targets for the treatment of cancer. The telomerase enzyme is in charge of maintaining the telomeres, which cover and safeguard the ends of chromosomes.
Cells lacking telomerase experience replicative senescence, a growth stop, after a certain number of cell divisions. Inactivating essential cell cycle checkpoints is the first step in the vast majority of malignancies to overcome replicative senescence. Once telomeres are critically short, telomerase expression is then increased.
It has been demonstrated that telomerase suppression causes telomere shortening in cancer cells. After enough cell divisions, this attrition might result in telomere uncapping and the activation of apoptosis.
To learn more about telomerase click here
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