Answer:
Parathyroid (PTH)
Explanation:
In the kidney, parathyroid hormone (PTH) blocks reabsorption of phosphate in the proximal tubule while promoting calcium reabsorption in the ascending loop of Henle, distal tubule, and collecting tubule. Parathyroid hormone (PTH) promotes absorption of calcium from the bone in 2 ways.
Answer:the 1st one is 2 1/2 ml the 2nd is um 1ml the 3rd is 4 1/4ml and the 5th is
23ml
Explanation:
Awareness context does the nurse determine this is closed awareness
<h3>What is pancreatic cancer ?</h3>
However, as the pancreatic cancer progresses and spreads, upper abdominal pain frequently begins to manifest, occasionally moving to the back. Following a meal or lying down, the pain might get worse. Jaundice, nauseousness, appetite loss, weight loss, exhaustion, sluggishness, and depression are some additional symptoms that could exist.
- Known risk factors for pancreatic cancer include smoking, diabetes, chronic pancreatitis, or inflammation of the pancreas, a family history of the disease, and a few genetic syndromes. Another contributing factor might be carrying extra weight that is unhealthy for your body.
Learn more about Pancreatic cancer here:
brainly.com/question/28217851
#SPJ4
Answer: $25,434.12
Explanation:
Gross income refers to the sum of all the salaries, wages, interest payments, rents, profits, and every other forms of earnings, before tax is removed.
Since the first Income earned is $15,667.88 while the second income is $9766.24, then Kyra's gross income from the two jobs will be:
= $15667.88 + $9766.24
= $25434.12
Answer:
Okay
Explanation:
Human topoisomerase I plays an important role in removing positive DNA supercoils that accumulate ahead of replication forks. It also is the target for camptothecin-based anticancer drugs that act by increasing levels of topoisomerase I-mediated DNA scission. Evidence suggests that cleavage events most likely to generate permanent genomic damage are those that occur ahead of DNA tracking systems. Therefore, it is important to characterize the ability of topoisomerase I to cleave positively supercoiled DNA. Results confirm that the human enzyme maintains higher levels of cleavage with positively as opposed to negatively supercoiled substrates in the absence or presence of anticancer drugs. Enhanced drug efficacy on positively supercoiled DNA is due primarily to an increase in baseline levels of cleavage. Sites of topoisomerase I-mediated DNA cleavage do not appear to be affected by supercoil geometry. However, rates of ligation are slower with positively supercoiled substrates. Finally, intercalators enhance topoisomerase I-mediated cleavage of negatively supercoiled substrates but not positively supercoiled or linear DNA. We suggest that these compounds act by altering the perceived topological state of the double helix, making underwound DNA appear to be overwound to the enzyme, and propose that these compounds be referred to as ‘topological poisons of topoisomerase I’