<span>adenine and guanine i think</span>
Answer and Explanation:
The mechanism of action of statins begins with the inhibition of a protein called HMG-CoA reductase, allowing its substrate to accumulate in the body and promote cholesterol reduction. At that moment, a transporter called OATP (Organic Anion Transporting Polypeptides) takes statins to the epatic tissue where they will be transformed and metabolized until they are eliminated through bile. This elimination is influenced by the P-glycoprotein, which is the main cause of the efflix presented by the bili. This process can get help from other molecules like some protease inhibitors, digoxin and cyclosporine.
Mendel was able to attribute the variation observed in the offspring of his experiment to the controlled fertilization process.
Mendel was able to control pollination and, thus, the fertilization process in the pea plants used for his experiment.
Mendel was able to effectively predict the outcome with self-pollination or cross-pollination with different results coming from each. Thus, he logically concluded that the variation observed in the offspring of his crosses is due to the fact that he controlled the fertilization process.
More on Mendel's experiments can be found here: brainly.com/question/3186121?referrer=searchResults
The teratogens can be defined as any physical, chemical, or infections agents that have the ability to penetrate the protective layers of the embryo, and affect its viability. The overall development and the development of the different organ systems might be affected because of these teratogens. When the embryo is exposed to the teratogens after the organogenesis is complete, it is most likely that the fetus will suffer from the stunted growth. When the organogenesis is complete, the organs are already formed, and hence, no severe damage can occur to them. Down's syndrome will result if the chromosomal abnormality occur in the fetus, which is not likely after the organogenesis is already complete. Any kind of severe anatomical defects are also least likely after organogenesis is completed. Therefore, the chances of having stunted growth are high.
I just got this question on my test, the answer is D) glycogen. All excess glucose is stored long-term in the liver and muscle cells as glycogen when animals need it for energy.
