Step 1: Isolate the two kinds of DNA.
Step 2: Treat the plasmid and foreign DNA with the same restriction enzyme.
Step 3: Mix the foreign DNA with chopped plasmids.
Step 4: Add DNA ligase.
I presume your question is, how many chromosomes would be found in the resulting daughter cells of meiosis?
If parent cell has 16 chromosomes, the first division in Meiosis of the cell will result in 8 chromosomes, and the second division (I.e. the separation of the sister chromatids of the 8 chromosomes) results in 8 daughter chromosomes.
The number of chromosomes in each cell resulting from meiosis is 8.
Hope this helps! :)
Answer:
True
Explanation:
Molecules always move faster when heated up.
Answer:
Dominant allele does not completely conceal recessive allele.
Snapdragon with genotype Rr (R being red and r being white), would have a phenotype of pink flowers.
Explanation:
Incomplete dominance is where a dominant allele is not able to completely conceal a recessive allele, usually leading to a phenotype which appears to be a combination of the two.
For example, in snapdragons:
The allele for red flowers (R) is dominant over the allele for white flowers (r). Let's say a snapdragon flower had the genotype Rr, one allele for red flowers and one for white. In the case of 'normal' dominance the dominant red flower allele (R) would mask the effects of the recessive white flower allele (r), resulting in the phenotype (outward observable characteristics) of having red flowers.
However here in the case of incomplete dominance, the dominant allele would not be able to fully cover up the effects of the white flower allele, meaning that both colors (red and white) are expressed in the phenotype, resulting in pink flowers.
Hope this helped!
Answer: pathogen–host coevolution
Explanation:
A major driver of evolution is Reciprocal coevolution between host and pathogen. Rather than pathogen, one-sided adaptation to a nonchanging host, high virulence specifically favoured during pathogen–host coevolution. In all of the independent replicate populations under coevolution, the pathogen ( B. thuringiensis ) genotype BT-679 with known nematocidal toxin genes of C. elegans and high virulence specifically swept to fixation but only some of them go under one-sided adaptation,
so relative change in B. thuringiensis virulence was greater than the relative change in C. elegans resistance is due to the elevated copy numbers of the plasmid containing the nematocidal toxin genes
.
