Answer:
Sickle cell anemia refers to a recessive inheritance disorder, in which every individual carries two copies of a gene. In normal individuals, both the copies of the globin gene are normal, and they generate 2 and 4 kb fragments of DNA on digestion by BseR1. A patient with sickle cell anemia is homozygous for the recessive gene and exhibits a mutation in both the copies of a globin gene.
Therefore, BseR1 could not digest the globin gene and generate a single 6Kb fragment of DNA. In a patient acting as a carrier, one copy of a globin gene is mutated and the other is normal. As a consequence, BseR1 digestion generates 6kb of the fragment of DNA (mutated copy) and 4 and 2 kb from the normal copy of a gene.
While waiting for a signal to contract, a muscle cell membrane maintains a slightly higher positive charge on the outside only because sodium-potassium ATPase pumps are available to generate this difference.
Only because sodium-potassium ATPase pumps are available to provide this difference, a muscle cell membrane maintains a marginally higher positive charge on the outside while waiting for a signal to contract.
To produce an action potential, these sodium pumps can transfer sodium ions across a cell's membrane. The body's muscles can then contract as a result of electrical signals travelling through it.
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The priority nursing intervention for this client is FOR THE NURSE TO TAKE CARE OF THE IMPAIRED GAS EXCHANGE.
Dyspnea is a medical condition in which the affected individuals have difficulty in breathing properly, that is, such individuals usually experience shortness of breath. Shortness of breath is one of the symptoms that are associated with heart failure. In the scenario given above, the first thing for the nurse to do is to ensure that the patient is breathing normally again, before setting out to take care of other things.
