Answer:
There is no cure for cystic fibrosis, but treatment can ease symptoms and reduce complications.
Explanation:
1). For those with cystic fibrosis who have certain gene mutations, doctors may recommend a newer medication called ivacaftor. This medication may improve lung function and weight, increases the activity of Cystic fibrosis transmembrane conductance regulator (CFTR)protein and reduce the amount of salt in sweat. It has been approved by the Food and Drug Administration for people with cystic fibrosis who are age 6 and older. The dose depends on your weight and age.
2). For people with a certain gene mutation who are age 12 and older, another drug is available that combines ivacaftor with a medication called lumacaftor. This drug is called orkambi.
The use of Orkambi may improve lung function and reduce the risk of exacerbations.
I hope you're clear on this Daxxy
Answer:
<em>Like every other organism the population size of the humans have increased over time.</em>
Explanation:
With the improvements in science and technology, life becomes better and better for human beings. In the past, when there were no discoveries for curing diseases, the human population declined because of the spread of various diseases and famine conditions.
But as the developments in the scientific world are made, it has been easier to cure diseases and other problems. As a result, the population size of the human goes on increasing.
When a DNA sequence alteration results in a stop codon rather than a codon that specifies an amino acid, it is known as a nonsense mutation. This is rarely seen in 10% of patients with genetic disease.
<h3>What is nonsense mutation?</h3>
A nonsense mutation in a DNA sequence causes a premature stop codon, also known as a nonsense codon, in the transcribed mRNA as well as a shortened, ineffective, and typically nonfunctional protein product.
Because stop codons, also known as nonsense codons, signal the completion of protein synthesis rather than encoding for an amino acid, they are the source of the term "nonsense mutation."
Examples of illnesses for which nonsense mutations have been implicated as contributing factors include: Cystic fibrosis (produced by the G542X mutation in the cystic fibrosis transmembrane conductance regulator); (CFTR) Beta-globin (thalassemia) Hurler disease.
Learn more about nonsense mutation, here:
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Answer: A
Explanation: the ice age posed a new chalenge to deer mice and on the ice the preadators could spot the darker deer mice better than the light deer mice.