Regulation of HMG-CoA reductase is termed as the rate of controlling enzyme of the mevalonate pathway which produces other isoprenoid and cholesterol.
The regulation of cholesterol is done by regulating the rate which determines step catalyzed by HGM -CoA reductase.
There is long-term and short-term regulation where HGM-CoA is inhibited by phosphorylation and catalyzed of proteins. There is regulated proteolysis of HGM-CoA reductase where degradation of HMG-CoA reductase by oxidized derivatives of cholesterol.
Answer:
C
Explanation:
I believe this is the answer.... UWU
Answer:
Neutrophils help fight infections because they ingest microorganisms and secrete enzymes that destroy them. A neutrophil is a type of white blood cell, a type of granulocyte and a type of phagocyte.
Explanation:
Neutrophils display adhesion glycoproteins on their surface to bind endothelial and subendothelial structures. They move randomly until they find a damaged site. Unless neutrophils are activated, endothelial cells do not tend to adhere. When inflammation mediators (IL-1, FNT) activate endothelial cells, they express P-selectin and E-selectin on the surface. The expression of glycoproteins and L-selectin cause the initial adhesion of the non-stimulated neutrophil to the activated endothelium, slowing it down by rolling it over the endothelium. Activated endothelial cells, opsonized particles, immune complexes, FEC-G, FEC-GM and chemoattractants produce factors that stimulate neutrophil activation. Expressing β2 integrin (endothelium adhesion molecule) Neutrophils expand and form pseudopods. Neutrophil activation also promotes degranulation, superoxide generation, and arachidonate metabolite production.
