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trasher [3.6K]
3 years ago
5

A premature infant develops jaundice. Laboratory test results are negative for hemolytic disease of the newborn, but the infant’

s bilirubin level continues to rise. Abnormal urinalysis test results include a dark yellow color, positive Ictotest, and needle-shaped crystals seen on microscopic examination. What is the most probable cause of the infant’s jaundice?
Medicine
1 answer:
DENIUS [597]3 years ago
7 0

Answer:

Patient has some kind of congenital disease that causes jaundice.

You might be interested in
an infant born with esophageal atresia and tracheoesophageal fistual receives a prescription for enternal feedings after correct
earnstyle [38]

An infant born with esophageal atresia and tracheoesophageal fistula receives a prescription for internal feedings after corrective surgery. <u>An infant is born with esophageal atresia and tracheoesophageal fistula.</u>

<u />

Esophageal atresia is a beginning defect in which a part of a baby's esophagus (the tube that connects the mouth to the stomach) no longer increases well. Esophageal atresia is a start defect of the swallowing tube (esophagus) that connects the mouth to the belly.

The precise cause of EA remains unknown, but it appears to have some genetic additives. as much as 1/2 of all infants born with EA have one or greater other beginning defects, together with: trisomy thirteen, 18, or 21. other digestive tract problems, such as intestinal atresia or imperforate anus.

Oesophageal atresia is a concept to be because of trouble with the development of the esophagus even as the child is in the womb, although it's not clear exactly why this takes place. The condition is extra, not unusual in babies of mothers who had too much amniotic fluid in being pregnant (polyhydramnios).

Learn more about Esophageal atresia  here:

brainly.com/question/28256726

#SPJ4

6 0
1 year ago
Explain how neurons communicate. Include a description of the action potential and how the action potential is converted into a
suter [353]

Answer:

Action potentials and chemical neurotransmitters.

Explanation:

Neurons communicate with each other via electrical events called ‘action potentials’ and chemical neurotransmitters.  At the junction between two neurons (synapse), an action potential causes neuron A to release a chemical neurotransmitter.  The neurotransmitter can either help (excite) or hinder (inhibit) neuron B from firing its own action potential.

In an intact brain, the balance of hundreds of excitatory and inhibitory inputs to a neuron determines whether an action potential will result.  Neurons are essentially electrical devices. There are many channels sitting in the cell membrane (the boundary between a cell’s inside and outside) that allow positive or negative ions to flow into and out of the cell.  Normally, the inside of the cell is more negative than the outside; neuroscientists say that the inside is around -70 mV with respect to the outside, or that the cell’s resting membrane potential is -70 mV.

This membrane potential isn’t static. It’s constantly going up and down, depending mostly on the inputs coming from the axons of other neurons. Some inputs make the neuron’s membrane potential become more positive (or less negative, e.g. from -70 mV to -65 mV), and others do the opposite.

These are respectively termed excitatory and inhibitory inputs, as they promote or inhibit the generation of action potentials (the reason some inputs are excitatory and others inhibitory is that different types of neuron release different neurotransmitters; the neurotransmitter used by a neuron determines its effect).

Action potentials are the fundamental units of communication between neurons and occur when the sum total of all of the excitatory and inhibitory inputs makes the neuron’s membrane potential reach around -50 mV (see diagram), a value called the action potential threshold.  Neuroscientists often refer to action potentials as ‘spikes’, or say a neuron has ‘fired a spike’ or ‘spiked’. The term is a reference to the shape of an action potential as recorded using sensitive electrical equipment.

Neurons talk to each other across synapses. When an action potential reaches the presynaptic terminal, it causes neurotransmitter to be released from the neuron into the synaptic cleft, a 20–40nm gap between the presynaptic axon terminal and the postsynaptic dendrite (often a spine).

After travelling across the synaptic cleft, the transmitter will attach to neurotransmitter receptors on the postsynaptic side, and depending on the neurotransmitter released (which is dependent on the type of neuron releasing it), particular positive (e.g. Na+, K+, Ca+) or negative ions (e.g. Cl-) will travel through channels that span the membrane.

Synapses can be thought of as converting an electrical signal (the action potential) into a chemical signal in the form of neurotransmitter release, and then, upon binding of the transmitter to the postsynaptic receptor, switching the signal back again into an electrical form, as charged ions flow into or out of the postsynaptic neuron.

4 0
3 years ago
When drowning is suspected as the cause of cardiac arrest in a child or infant, deliver 2 initial ventilations before starting c
vitfil [10]

Answer:

true

Explanation:

i got my answer from american red cross  also my gu went with true sorry if wrong

6 0
2 years ago
Explain how and why drinking water affects:<br> (A) Blood plasma<br> (B) Blood plasma osmolarity
Oxana [17]

Answer:

(A) - The volume of the blood plasma increases on overhydration and decreaes on dehydration.

(B) - Osmolality of blood increases with the dehydration and decreases with the overhydration.

Explanation:

If the levels of drinking water is increased, then the plasma becomes more dilute and the plasma volume increases because <u>the water will be reabsorbed more and which as a result reduces plasma concentration of the solutes and increases volume of plasma.</u>

Plasma osmolality measures electrolyte-water balance of the body. <u>As mentioned, above on overhydration, the plasma concentration of the solutes decraeses which also decreases osmolality. So, Osmolality of blood increases with the dehydration and decreases with the overhydration. </u>Increased osmolality in blood stimulate the secretion of antidiuretic hormone which results in the increased water reabsorption and therefore, more concentrated urine and hence blood plasma with less concentration.

4 0
3 years ago
Which hormone would not be severely affected by damage to hypothalamus
WARRIOR [948]

Answer:

Melatonin

Melatonin is produced by the pineal gland in the brain.

Explanation:

Hormones of the hypothalamus include

oxytocin

prolactin

Thyroid stimulating Hormones

4 0
3 years ago
Read 2 more answers
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