All of the above points are valid for fossils' contributions in understanding the process of evolution. They allow us to study the ancestry, we can see the homologous organs or structures, if fossils are well-preserved; different fossils can lead us to follow the cycles of the changes that occurred during macroevolutions, and fossils can be used to study different populations which had different body structures for surviving in different environmental conditions.
Chromatin
Chromosome, the highest packing level of DNA, is packed in nucleus. DNA strand itself is packed in chromatin. Chromatin is DNA packaging into 30nm fibers that involve DNA strand with scaffolding around DNA proteins, like histones, in eukaryotic cells’ nucleus.
Explanation:
The DNA strand is folded into nucleosomes, bead-like structures on the DNA and fold up into the 30nm fibers. During meiosis and mitosis, the chromatin is super-coiled up into thicker fibers called chromosomes. Chromosomes within the nucleus can be viewed by a light microscope in a cell in metaphase.
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Numerous degenerative neurological conditions, most notably Parkinson's disease, have been linked to an excessive buildup of alpha synuclein (a-syn) in the brain. Intraneuronal inclusions, often known as Lewy bodies, are neuropathological characteristics seen in Parkinson's disease, Lewy body dementia, and other synucleopathies. The aggregation of a-syn is their main structural component. A-syn accumulation, aggregation, and ensuing Lewy body formation can be attributed to a variety of biological processes. These include genetic changes in parkin, synuclein, or the deubiquitinating enzyme ubiquitin C-terminal hydrolase (UCH-L1), which results in less efficient removal of a-syn via the ubiquitin proteasomal pathway (UPP). Additionally, environmental variables and an age-related decline in antioxidant defense mechanisms that heighten oxidative stress and can have an impact on the formation or clearance of a-syn are intracellular insults.
We focused on changes in the aggregation and clearance of a-syn as impacted by the UPP and the oxidative stress pathways in our dynamic models of a-syn processing in both normal and various disease states. A free radical profile similar to that observed in vivo after exposure to the neurotoxin 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine is produced during simulation of enhanced oxidative stress (MPTP). To replicate the kinetics of a-syn that correlates to the neuropathology reported for the sporadic and hereditary types of Parkinson's disease, different model parameters of oxidative stress, UPP failure, or both routes are used. With the use of this in silico model, it is possible to evaluate the kinetics of pathway elements and more accurately identify and validate key pharmaceutical targets.
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Answer:
anything that could escalate
Explanation:
what are the choices? i would think that you keep your temper very low do not raise your voice and try to get them into a seperate room so that you can try to calm them down
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