Answer:
Some bacteria due to their structures and physiology are inherently resistant to some antibiotics. This could be due to lacking transport mechanism or lacking receptor sites. Bacterium develop resistance mechanisms by using instructions provided by their DNA. Resistance genes are often found in plasmids which are small pieces of DNA that carry genetic instructions from one germ to another. Meaning that some bacteria can share their DNA and cam make other germs become resistant.
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Temperature affects spermatogenesis, which functions best at body temperatures just a little lower than those.
<h3>Abstract:</h3>
To keep testicular temperatures below those of the body core, adequate thermoregulation is essential. The process of mammalian spermatogenesis and the resulting spermatozoa are negatively impacted by elevated testicular temperature. Therefore, sperm quality can be affected and the likelihood of infertility is increased by thermoregulatory dysfunction resulting in heat stress. This article reviews a variety of internal and external factors that may lead to testicular heat stress. We go into more detail on how heat stress affects the spermatogenesis process, the resulting epididymal spermatozoa, germ cells, and the alterations that result in the testis.
We also go over the chemical reactions of germ cells to heat exposure and potential processes, such as apoptosis, DNA damage, and autophagy, that could lead to heat-induced germ cell damage. Further explanation is provided for the intrinsic and extrinsic processes involved in the complex mechanism of germ cell death. These intricate apoptotic pathways ultimately result in the demise of germ cells.
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Answer:
A variety of genes are involved in the control of cell growth and division. Tight regulation of this process ensures that a dividing cell's DNA is copied properly, any errors in the DNA are repaired, and each daughter cell receives a full set of chromosomes.
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Answer:
Chloroplast of Leaf Cells
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