Wild Bedbugs become insecticide resistant because of the mutations and natural selections.
<h3><u>Explanation</u>:</h3>
As the huge amount of pesticides and insecticides are sprayed in the rooms for cleaning, the pests and insects like bedbugs dies in huge portions because of the toxin. But some of the bedbugs remain alive as they have mutations that help them to detoxify the toxins given, or bypass the metabolic processes so that the toxins don't hamper them much.
Now as the population becomes very small(bottle neck effect), the nature selects these organisms over the other to propagate more sufficiently and enormously. As the nutrients and supplies are also available, so the bedbugs don't suffer any lack of nutrition which can be a determining factor of their population.
Thus the wild bedbugs become resistant to insecticides while the experimental one remain succeptible to insecticides.
The highest level of organization for living things is the BIOSPHERE; it encompasses all other levels. The biological levels of organization of living things arranged from the simplest to most complex are: organelle, cells, tissues, organs, organ systems, organisms, populations, communities, ecosystem, and biosphere.
They both hunt for food and will ruthlessly kill you before you can cry, "MAMA!"
The right answer is glutamate
Glutamate is a major excitatory neurotransmitter associated with learning and memory.
It is also associated with Alzheimer's disease whose first symptoms are felt in memory.
Most people unknowingly consume monosodium glutamate (MSG), a commonly used food additive.
An experiment was done on healthy patients, giving them 12 grams of glutamate. It was found that the experience led to headaches, nausea or jaw pain in 50-60% of cases.
It was also measured the level of glutamate contained in the muscles of patients with jaw problems. The tests concluded that the most painful muscles were those containing the highest levels of glutamate.
According to him, one of the keys to chronic pain could lie in the development of drugs that reduce the rate of glutamate.
