Well Stacy I think it may be able to eat those plants without dying
Which signal are you talking about?
It is B because I had to do this question to
Answer:
B) a nonsense mutation; this is because a nonsense mutation results in the change of a regular amino acid codon into a stop codon, which ceases translation. This fits with the problem's description of the protein that causes the symptoms as too short, as translation is the process by which proteins/polypeptides are created. A missense mutation would not be the answer because it still codes for an amino acid, which would not shorten the protein. A duplication of the gene would probably just lengthen the protein or not affect its length at all.
<span>Protein</span>
<span>Cilia and flagella are made up of microtubules covered by
plasma membrane and they projection from the cell. Thus, cilia are flagella are
motile and designed to move substances over or around the cell and they can as
well move the cell itself. In eukaryotes, they are conserved in their polypeptide
composition, structure and function as motile and sensory organelles.</span>
