Question 1:
Exons are gene fragments whose DNA sequence after transcription is found in mature mRNAs. This part of the gene is most often coding.
The size of the exons in the genomes follows a log-normal distribution, with an average length of about 150 nucleotides, knowing that in eukaryotes, each gene contains several exons and introns (an average of 8) so the size is 8*150 = 1200 bp.
Question 2:
Introns are fragment of a gene located between two exons. Introns are present in immature mRNA and absent in mature mRNA. "Non-coding" fragment of the gene.
The introns average in a gene is 3365 bp including 3'UTR and 5'UTR and intermediate introns.
2. In 1945 a U.S. Air Force bomber accidentally crashed into the Empire State Building, killing fourteen people and damaging the 78th and 79th floors.
4. No infinitives are used. Sentence 4 contains a gerund.
5. includes Steve writing about himself in the first person.
That what i got and if any incorrect then sorry I try my best.
It forms in lagoons where ocean waters high in calcium and sulfate content can slowly evaporate and be regularly replenished with new sources of water. The result is the accumulation of large beds of sedimentary gypsum. Gypsum is commonly associated with rock salt and sulfur deposits.
I have the answer to dead zone hoped it helped you in some way!☺❤
<h2>CRISPR/Cas9</h2>
Explanation:
CRISPR can be used to reintroduce dystrophin back into the KO mouse
- CRISPR stands for Clustered Regularly Interspaced Short Palindromic Repeats and is used to for gene editing
- CRISPR/Cas-mediated genome editing has been shown to permanently correct DMD mutations and restore dystrophin function in mouse models
- Germline editing by injecting zygotes with CRISPR/Cas9 editing component was first done in mdx mice by correcting the mutated exon 23
- Postnatal editing of mdx mice was then achieved using recombinant adeno-associated virus to deliver CRISPR/Cas9 genome editing components and correct the dystrophin gene by skipping or deleting the mutated exon 23 in vivo
- Germline and postnatal CRISPR/Cas9 editing approaches both successfully restored dystrophin function in the mice and same technique can be used for KO mouse model
